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Schonig, K.; Schwenk, F.; Rajewsky, K.; Bujard, H. Nucleic Acids Res., 2002, 30, e134. Metzger, D.; Clifford, J.; Chiba, H.; Chambon, P. Proc. Natl. Acad. Sci. USA, 1995, 92, 6991-6995. Schneider, A.; Zhang, Y.; Guan, Y.; Davis, L.S.; Breyer, M.D. J. Physiol. Renal Physiol., 2003, 284, F411-417. Bockamp, E.; Maringer, M.; Spangenberg, C.; Fees, S. Physiol. Genomics., 2002, 11, 115-132. Lamartina, S.; Roscilli, G.; Rinaudo, C.D.; Sporeno, E.; Silvi, L.; Hillen, W.; Bujard, H.; Cortese, R.; et al. Hum. Gene Ther., 2002, 13, 199-210. Kistner, A.; Gossen, M.; Zimmermann, F.; Jerecic, J. Proc. Natl. Acad. Sci. USA, 1996, 93, 10933-10938. Baron, U.; Gossen, M.; Bujard, H. Nucleic Acids Res., 1997, 25, 2723-2729. Gossen, M.; Bujard, H. Proc. Natl. Acad. Sci. USA, 1992, 89, 5547-5551. Moody, S.E.; Sarkisian, C.J.; Hahn, K.T.; Gunther, E.J.; Pickup, S.; Dugan, K.D.; Innocent, N.; Cardiff, R.D.; et al. Cancer Cell, 2002, 2, 451-461. Baasner, S.; von Melchner, H.; Klenner, T.; Hilgard, P.; Beckers, T. Oncogene, 1996, 13, 901-911. Giavazzi, R.; Sennino, B.; Coltrini, D.; Garofalo, A.; Dossi, R.; Ronca, R.; Tosatti, M.P.; Presta, M. Am. J. Pathol., 2003, 162, 1913-1926. Liu, S.; Liu, W.; Jakubczak, J.L.; Erexson, G.L.; Tindall, K.R.; Chan, R.; Muller, W.J.; Adhya, S.; et al. Proc. Natl. Acad. Sci. USA, 2002, 99, 3770-3775. Giavazzi, R.; Giuliani, R.; Coltrini, D.; Bani, M.R.; Ferri, C.; Sennino, B.; Tosatti, M.P.; Stoppacciaro, A.; et al. Cancer Res., 2001, 61, 309-317. Hogan, B.; Beddington, R.; Costantini, F.; Lacy, E. Manipulating the mouse embryo. New York: Cold Spring Harbor Laboratory Press; 1994. Joyner, A. Gene targeting- A practical approach. Edited by Hames BD. New York: Oxford University Press; 1999. Eggan, K.; Rode, A.; Jentsch, I.; Samuel, C.; Hennek, T.; Tintrup, H.; Zevnik, B.; Erwin, J.; et al. Nat. Biotechnol., 2002, 20, 455-459. Seibler, J.; Zevnik, B.; Kuter-Luks, B.; Andreas, S.; Kern, H.; Hennek, T.; Rode, A.; Heimann, C.; et al. Nucleic Acids Res., 2003, 31, e12. Zambrowicz, B.P.; Sands, A.T. Nat. Rev. Drug Discov., 2003, 2, 38-51. Ihle, J.N. Cell, 2000, 102, 131-134. Jonkers, J.; Berns, A. Nat. Rev. Cancer, 2002, 2, 251-265. Lewandoski, M. Nat. Rev. Genet, 2001, 2, 743-755. Tronche, F.; Kellendonk, C.; Reichardt, H.M.; Schutz, G. Curr. Opin. Genet Dev., 1998, 8, 532-538. Reichardt, H.M.; Tronche, F.; Bauer, A.; Schutz, G. Biol. Chem., 2000, 381, 961-964. Schmid, W.; Cole, T.J.; Blendy, J.A.; Schutz, G. J. Steroid Biochem. Mol. Biol., 1995, 53, 33-35. Soriano, P. Nat. Genet., 1999, 21, 70-71. Srinivas, S.; Watanabe, T.; Lin, C.S.; William, C.M.; Tanabe, Y.; Jessell, T.M.; Costantini, F. BMC Dev. Biol., 2001, 1, 4. Muyrers, J.P.; Zhang, Y.; Benes, V.; Testa, G.; Ansorge, W.; Stewart, A.F. EMBO Rep., 2000, 1, 239-243. Zhang, Y.; Muyrers, J.P.; Testa, G.; Stewart, A.F. Nat. Biotechnol., 2000, 18, 1314-1317. Waskow, C.; Paul, S.; Haller, C.; Gassmann, M.; Rodewald, H. Immunity 2002, 17, 277-288. Blume-Jensen, P.; Jiang, G.; Hyman, R.; Lee, K.F.; O'Gorman, S.; Hunter, T. Nat. Genet., 2000, 24, 157-162. Kissel, H.; Timokhina, I.; Hardy, M.P.; Rothschild, G.; Tajima, Y.; Soares, V.; Angeles, M.; Whitlow, S.R.; et al. EMBO J., 2000, 19, 1312-1326.
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RNA was treated with DNaseI Amersham Pharmacia Biotech ; . Complementary DNA cDNA ; was synthesized by the Moloney Murine Leukemia Virus M-MLV ; reverse transcriptase RT ; Life Technologies, Inc. ; with HSVtk gene specific primer, Ex4TKA, in 20 l of reaction mixture 33 ; . Nested PCR was performed. First, 20 cycles of PCR amplification were performed using 2 l of cDNA as template with 20 pmol primers, Ex3ATGS and Ex4TKA, in 50 l of reaction mixture containing 0.5 U Taq DNA polymerase, 50 mm KCl, 10 mm Tris-HCl, 0.1% Triton X-100, 0.2 mm dNTPs, 2.5 mm MgCl2 and 10% dimethylsulfoxide. Then 20 cycles of PCR amplification were performed using 2 l of the first PCR mixture as template with primers Ex3ATGSin and Ex4TKAin. Each amplification cycle consisted of denaturation at 94 C for 1 min, annealing at 55 C for 1 min, and extension at 72 C for 1.5 min. Nested PCR was also performed using serial diluted plasmid 250 to 250 16 6 pg ; template to show that PCR amplification was in the linear range. PCR products were analyzed in 2% agarose gels stained with ethidium bromide. Primers used for PCR amplification: Ex4TKA: 5 -GTGTAGATGTTCGCGATTGT-3 Ex3ATGS: 5 -ATGGTGCAGGACTATGTGCA-3 Ex3ATGSin: 5 -ATGAAGGCCAGTGAGCTGGA-3 Ex4TKAin: 5 -GGTACGTAGACGATATCGTC-3 and ceftin.
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SECTION III SELECTION OF DRUGS Section III.A--Selection of Drugs for Pneumococcal Infections Streptococcus pneumoniae is an alpha hemolytic gram-positive coccus that colonizes the nasopharynx of many children and some adults, especially in winter months and during viral infections. It accounts for at least one-third of acute otitis media and acute sinusitis cases, which makes it the most prevalent pathogen of the upper respiratory tract. It is also the one most likely to cause persistent infections that fail response to time and treatment ; and to cause serious, invasive complications of those infections, such as mastoiditis, bacteremia, and meningitis. Historically, pneumococci have been very sensitive to and easily treated with--any of the penicillins amoxicillin being most potent ; , macrolides erythromycin ; , cephalosporins, clindamycin, etc. Drug choices for penicillin-susceptible S. pneumoniae: Primary: Penicillin V Amoxicillin Amoxicillin clavulanate Augmentin ; if Hemophilus influenzae or M. catarrhalis might be present ; Alternatives: Erythromycin or clarithromycin Biaxjn ; or telithromycin Ketek ; or clindamycin plus sulfonamide with any above if H. influenzae or M. catarrhalis is likely ; Cefpodoxime Vantin ; or equivalents page 5 ; "Respiratory quinolones" page 16, Section I.I ; Levo-, gati-, or moxifloxacin. ATACAND .12 ATACAND HCT.12 atenolol .12 atenolol - chlorthalidone .12 ATROVENT.18 AUGMENTIN XR.7 AVALIDE.12 AVANDAMET .10 AVANDIA .10 AVAPRO .12 AVELOX.7 AVODART .16 azathioprine .17 AZMACORT .18 AZOPT.18 B baclofen.19 BACTROBAN .15 belladonna - phenobarbital .15 benazepril HCL.12 benazepril HCL-HCTZ .12 BENICAR .12 BENICAR HCT.12 benztropine mesylate.9 betamethasone dipropionate .15 betamethasone dp augmented.15 betaxolol HCL.12 bethanechol chloride.16 BIAXIN.7 BIAXIN XL .7 bisoprolol fumarate.12 bisoprolol fumarate HCTZ.12 brimonidine tartrate.18 budeprion SR.8 bumetanide .12 bupropion HCL .8 buspirone HCL.10 C and celexa. CD-ROM resources G Cochrane Controlled Trials Register CCTR ; 2002: Issue 1 ; searched: 2 April 2002 ; G Cochrane Database of Systematic Reviews CDSR ; 2002: Issue 1 ; searched: 2 April 2002 ; G EMBASE 1980February 2002 ; searched: 27 March 2002 ; G MEDLINE 1966March 2002 ; searched: 26 March 2002 ; G National Research Register NRR ; 2002: Issue 1 ; searched: 2 April 2002 ; G PREMEDLINE up to 22 March 2002 ; searched: 26 March 2002 ; G PsycINFO 1967week 3, July 2002 ; searched: 3 September 2002 ; Online resources searched 8 April 2002 ; G Conference Papers Index CPI ; 1973 onwards ; Paper resources G Clinical evidence: a compendium of the best available evidence for effective health care. Issue 6, 2001. London: BMJ Publishing Group. The Joint Commission and National Quality Forum are now accepting nominations for the 2005 John M. Eisenberg Patient Safety and Quality Awards, which recognize individuals and healthcare organizations that are making significant contributions to improving healthcare quality and patient safety. The deadline for submissions is May 27, 2005. Nomination forms are available at qualityforum . Questions about the awards may be directed to EisenbergAward jcaho or info qualityforum and cephalexin and biaxin, because bkaxin and alcohol.
Candidates for mutagenesis. This selection was influenced by neither residue being conserved in the 2-AR, a receptor at which these 1-AR selective antagonists possess markedly reduced affinity 10, 000-fold lower ; . Here we unexpectedly report that, although neither mutation altered the pharmacological phenotype with regard to antagonist binding at the 1a-AR, these mutations had significant effects on the binding of catecholamine and phenethylamine agonists at this receptor, agonists for which all receptor contacts were thought to have been identified. Since these phenylalanine residues are not conserved in 2-ARs and -ARs, these results stress the differences in the agonist binding pocket of adrenergic receptors despite the fact they all bind the same endogenous ligands.

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