A in the case of products for human use, by persons holding an appropriate manufacturing licence granted by the Minister for Health. b in the case of products for animal use, by persons holding an appropriate manufacturers' licence granted by the National Drugs Advisory Board.
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BENZODIAZEPINES Treatment of anxiety Alprazolam Xanax ; Bromazepam Lectopam ; Chlordiazepoxide Apo-chlordiazepoxide ; Clorazepate Apo-Clorazepate ; Diazepam Valium ; Lorazepam Ativan ; Oxazepam Apo-Oxazepam ; Treatment of insomnia Flurazepam Dalmane ; Midazolam Apo-Midazolam ; Nitrazepam Mogadon ; Temazepam Restoril ; Triazolam Halcion ; Anticonvulsants Clobazam Frisium ; Clohazepam Rivotril ; TRICYCLIC ANTIDEPRESSANTS Amitriptyline Apo-Amitriptyline ; Clomipramine Anafranil ; Desipramine Norpramine ; Doxepine Sinequan ; Imipramine Tofranil ; Maprotiline Novo-Maprotiline ; Nortriptyline Aventyl ; Trimipramine Surmontil ; OPIOIDS Natural opioids Opium Morphine: Kadian, Morphine HP, M-Eslon, M.O.S., MS-Contin, MS-IR Codeine: Codeine, Codeine Contin, in association with numerous products e.g., Empracet ; Semi-synthetic opioids Buprenorphine: Buprenex United States ; , Subutex Europe ; Heroin: Diacetylmorphine, Diamorphine Synthetic opioids AGONISTS Alfentanil: Alfenta Diphenoxylate: Lomotil Fentanyl: Duragesic Hydrocodone: Hycodan Hydromorphone: Dilaudid, Hydromorph Contin Meperidine or Pethidine: Demerol Methadone: Metadol, Methadone Oxycodone: OxyContin, Oxy-IR, Percocet, Percodan, Supeudol Oxymorphone: Numorphan Propoxyphene: Darvon-N Remifentanil: Ultiva Sufentanil: Sufenta.
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1. Physician Drug & Diagnosis Audit, 1997 Report. Durham, NC: Quintiles; 1997. 2. Woods SW, Nagy LM, Kolesar AS, Krystal JH, Heninger GR, Charney DS. Controlled trial of alprazolam supplementation during imipramine treatment of panic disorder. J Clin Psychopharmacol. 1992; 12: 32-38. Dannon PN, Iancu I, Grunhaus LJ. Short-term Potentiation of Paroxetine With Clonaaepam in the Treatment of Patients With Panic Disorder: American Psychiatric Association Annual Meeting, Washington, DC, May 15-29, 1999. Washington, DC: American Psychiatric Association Press; 1999: 153. 4. American Psychiatric Association. Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition. Washington, DC: American Psychiatric Association; 1994. 5. First MB, Spitzer RL, Gibbon M, Williams JBW. Structured Clinical Interview for DSM-IV Axis I Disorders--Patient Edition SCID-I P, Version 2.0 ; . New York, NY: Biometrics Research Department, New York State Psychiatric Institute; 1995. 6. DiNardo PA, Brown TA, Barlow DH. Anxiety Disorders Interview Schedule: Lifetime Version ADIS-IV-L ; . Albany, NY: Phobia and Anxiety Disorders Clinic; 1994. 7. Sheehan DV, Lecrubier Y. MINI PLUS version 4.4, 1 21 rev ; . Tampa, Fla: University of South Florida; 1994. 8. Shear MK, Brown TA, Barlow DH, Money R, Sholomskas DE, Woods SW, Gorman JM, Papp LA. Multicenter collaborative panic disorder severity scale. J Psychiatry. 1997; 154: 1571-1575. Ballenger JC, Davidson JR, Lecrubier Y, Nutt DJ, Baldwin DS, den Boer JA, Kasper S, Shear MK. Consensus statement on panic disorder from the International Consensus Group on Depression and Anxiety. J Clin Psychiatry. 1998; 59 suppl 8 ; : 47-54. 10. Pollack MH, Otto MW, Worthington JJ, Manfro GG, Wolkow R. Sertraline in the treatment of panic disorder. Arch Gen Psychiatry. 1998; 55: 1010-1016. Barlow DH, Gorman JM, Shear MK, Woods SW. Cognitive-behavioral therapy, imipramine, or their combination for panic disorder: a randomized controlled trial. JAMA. 2000; 283: 2529-2536. Hamilton M. The assessment of anxiety states by rating. Br J Med Psychol. 1959; 32: 50-55. Guy W. ECDEU Assessment Manual for Psychopharmacology. Washington, DC: US Department of Health Education and Welfare; 1976. Publication 76-338. 14. Sheehan DV. The Anxiety Disease. New York, NY: Bantam Books; 1986. 15. Charney DS, Woods SW. Benzodiazepine treatment of panic disorder: a comparison of alprazolam and lorazepam. J Clin Psychiatry. 1989; 50: 418-423. Londborg PD, Wolkow R, Smith WT, DuBoff E, England D, Ferguson J, Rosenthal M, Weise C. Sertraline in the treatment of panic disorder: a multisite, doubleblind, placebo-controlled, fixed-dose investigation. Br J Psychiatry. 1998; 173: 54-60. Pohl RB, Wolkow RM, Clary CM. Sertraline in the treatment of panic disorder: a double-blind multicenter trial. J Psychiatry. 1998: 1189-1195. 18. Lavori P. ANOVA, MANOVA, My black hen. Arch Gen Psychiatry. 1990; 47: 775-778. Olsen C. On choosing a test statistic in multivariate analysis of variance. Psychological Bull. 1976; 83: 579-586. Ekstrom D, Quade D, Golden RN. Statistical analysis of repeated measures in psychiatric research. Arch Gen Psychiatry. 1990; 47: 770-772. Cohen J. Statistical Power Analysis for the Behavioral Sciences. 2nd ed. Hillsdale, NJ: Lawrence Erlbaum Associates; 1988. 22. Rickels K, Case WG, Schweizer E, Garcia-Espana F, Fridman R. Benzodiazepine dependence: management of discontinuation. Psychopharm Bull. 1990; 26: 63-68. Shear MK, Maser JD. Standardized assessment for panic disorder research: a conference report [review]. Arch Gen Psychiatry. 1994; 51: 346-354 and
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ESS-EMCH SECTION 12 Paediatric emergencies Last updated 10 5 2007 glucose- hypoglycaemia less than 2.5 mmol litre 45mg dl ; is an important cause of impaired consciousness in children. CNS function may be compromised by convulsions, drugs, and CNS infections CNS compromise presents with neurological deficit, and effects the respiratory and cardiovascular systems Neurological assessment Conscious level A rapid assessment of conscious level can be made by using the AVPU scoring system.
C. Analysis of Team Effectiveness The basic assumption for using a team decision making process is that a group t decision should be better than the decision of an average team member. Therefore, the role of the facilitator in assessing the teams' performance on completing a management questionnaire is to illustrate how the team decision compares with the decisions of the individual team members. The summary of team scores work sheet see Figure A-2, page 181 ; is used by the facilitator in making an assessment of the team's performance. After all teams have completed the scoring task see Figure A-1, page 179 ; , the facilitator prepares the summary of team scores table and writes it on the board and
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Selective Serotonin Reuptake Inhibitors 20mg, 40mg BCF Citalopram BCF Fluoxetine Paroxetine BCF Sertraline Tricyclics BCF Amitriptyline Desipramine Doxepin BCF Imipramine BCF Nortriptyline ANTIPARKINSON AGENTS Acetylcholinesterase Inhibitor Donepezil Anticholinergic BCF Benztropine BCF Trihexyphenidyl Dopamine agonists Amantadine Bromocriptine BCF Carbidopa Levodopa, Immediate-Release ANTIPSYCHOTICS Chlorpromazine Haloperidol BCF Quetiapine BCF Risperidone Thioridazine ANXIOLYTICS HYPNOTICS BCF Buspirone CIV BCF Cl0nazepam 5mg, 10mg 1mg CIV BCF Diazepam BCF Doxepin BCF Hydroxyzine HCl BCF Hydroxyzine HCl CIV Lorazepam CIV Temazepam CNS STIMULANTS ADHD AGENTS CII Amphetamine Dextroamphetamine CII B Amphetamine Dextroamphetamine, CF Extended-Release CII Dextroamphetamine CII B CF Methylphenidate CII Methylphenidate CII B CF Methylphenidate, Extended-Release MIGRAINE AGENTS CIV Butalbital Acetaminophen Caffeine Ergotamine caffeine CIV Isometheptene Dichlorphenazone BCF Acetaminophen Rizatriptan, Disintegrating BCF Sumatriptan BCF Zolmitriptan BCF Zolmitriptan-ZMT Cyclophosphamide BCF Goserelin Hydroxyurea Megestrol BCF Methotrexate BCF Tamoxifen ANESTHETIC Proparacaine Tetracaine ANTIBACTERIALS Bacitracin Polymyxin B Ciprofloxacin BCF Erythromycin BCF Gentamicin BCF Gentamicin Levofloxacin BCF Neomycin Polymyxin B Bacitracin BCF Neomycin Polymyxin B Gramicidin BCF Sulfacetamide BCF Sulfacetamide Tobramycin Dexamethasone BCF Trimethoprim Polymyxin B ANTIHISTAMINE Olopatadine ANTIHISTAMINE VASOCONSTRICTOR Naphazoline Pheniramine Tetrahydrozoline CORTICOSTEROIDS BCF Prednisolone Acetate Fluorometholone GLAUCOMA AGENTS 0.1% 0.025-0.3% 0.05% 000 400 Units 1mg 100mg 65 Ophthalmic Soln Ophthalmic Soln Ophthalmic Oint Ophthalmic Soln Ophthalmic Oint Ophthalmic Soln Ophthalmic Oint Ophthalmic Soln Ophthalmic Oint Ophthalmic Soln Ophthalmic Soln Ophthalmic Oint Opthalmic Susp Ophthalmic Soln Ophthalmic Soln Ophthalmic Soln Ophthalmic Soln Ophthalmic Soln Ophthalmic Soln Alcaine Altacaine Polysporin Ciloxan Ilotycin Garamycin Garamycin Quixin Neosporin Neosporin Bleph-10 Bleph-10 Tobradex Polytrim Patanol Visine-A Visine Pred Forte FML 15ml bottle 15ml bottle 3.5 gram tube 5ml bottle 3.5 gram tube 5ml bottle 3.5 gram tube 5ml bottle 3.5 gram tube 10ml bottle 15ml bottle 3.5 gram tube 5ml bottle 10ml bottle 5ml bottle 15ml bottle 15ml bottle 5ml bottle 5ml bottle and
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Clonazepan is an alternate name for clonazepam.
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Also - i wait 4 hrs before taking the next drug don't know why - it just feels like the right amount of time.
The administration of clonazepam, rather than clobazam, could have had an impact on [Master B's] condition because the dose is 2-2.5 times that he would have received for his body weight and is likely to cause excessive sedation. Individuals who are heavily sedated have a risk of developing aspiration pneumonia if they are fed. It is likely that this did cause and contribute to his deterioration. Could the administration of clonazepam, rather than clobazam, have any long-term effects on [Master B's] well-being?.
Antidepressants Pharmacological investigations have demonstrated that OCD responds selectively to drugs that act as potent inhibitors of the synaptic reuptake of serotonin. The first medication demonstrated to be effective in OCD was clomipramine 150-250 mg day ; with 40% of patients versus 4% for placebo ; having a clinically significant decrease in symptoms independently of its antidepressant effect.157-159 Subsequently, all of the SSRIs have been shown to be effective, including fluvoxamine 100-300 mg day ; , fluoxetine 20-80 mg day ; , paroxetine 40-60 mg day ; , sertraline 50-200 mg day ; , and citalopram 2060 mg day ; .159 Most recent controlled trials find that about 50% of patients experience a 25% to 35% drop in scale scores of OCD, primarily utilizing the Yale-Brown Obsessive Compulsive Scale Y-BOCS ; . This magnitude of change typically results in significant improvement in function; however, interfering symptoms usually persist. Relative efficacy between the SRIs has been difficult to determine. Two meta-analysis suggested greater efficacy for chlorimipramine160, 161; however, these trials were performed over a 7- to 10-year time period, during which placebo rates rose significantly, making any conclusion suspect. In fact, in several head-to-head trials, clomipramine was found to have equal efficacy to fluoxetine, 162 paroxetine, 163 and sertraline, 164 with SSRIs being better tolerated than clomipramine. A more recent meta-analysis generally failed to find any significant difference between the SRIs, although it again suggested some advantage for clomipramine. However, this metaanalysis involved many of the trials mentioned above and has the same problem in interpretation.165 There was no observed difference in a trial comparing fluvoxamine, paroxetine, and citalopram.166 Due to their similar effects, it is difficult to choose between SSRIs, and the selection of a drug largely depends upon personal preference, even if the possibility of a drug interaction or the various pharmacokinetic profiles could influence the choice. Dosages of these medications have often been described as being significantly higher than antidepressant dosages eg, 60-80 mg day fluoxetine however, in large carefully controlled trials, there has been no observed significant difference between response to higher and lower dosages for the SSRIs eg, 50 and 200 mg day sertraline ; .167 This clinical impression may well relate to the slow onset of effectiveness with many patients taking 10 to 12 weeks to improve longer than 4-8 weeks for depression ; , during which physicians continue to raise the patients' doses, mistakenly thinking it was the increased dose, not time, that was responsible for improvement. For this reason, it is helpful to warn patients about this from the outset, and slowly titrate doses upwards to avoid side effects. Many patients will not respond or will partially respond to the first SSRI, but will respond to another antiobsessional agent. Therefore, sequential trials are frequently required, which easily can take up to a year to accomplish. Limited available evidence suggests that when effective pharmacotherapy is discontinued, most patients 90% ; do relapse.168 Therefore, current practice is to continue effective pharmacotherapy for at least 1 to 2 years or indefinitely. In a large extension study by Greist et al, 167 118 patients who had responded to 12 weeks' treatment with either sertraline or placebo continued their treatment, in a double-blind way, for 40 weeks. Therapy gains with sertraline were maintained with continued medication as long as they remained on active medication, without tolerance developing. The 59 patients who completed this study were followed up for a second year on open-label sertraline, whereupon they showed additional clinical improvements.169 Another trial with paroxetine demonstrated continued efficacy for 12 months in the majority of patients.170 The effectiveness of potent SRIs is now well established in the treatment of OCD, but despite these advances, nearly 40% to 60% of patients experience minimal to no improvement in symptoms with these treatments. Furthermore, in patients who do respond to SRIs, the degree of improvement is often incomplete, with few patients experiencing full symptom remission.171 For these reasons, attempts to augment or improve the average response with pharmacological strategies targeting serotonergic or other neurotransmitter systems are routine. There is no agent that is routinely effective as an augmenting agent, although there is some support for clonazepam, clonidine, trazodone, nefazodone, tryptophan, and pindolol.172 There is clear evidence of benefit for traditional neuroleptics173 and more recently the atypical neuroleptics eg, risperidone, olanzapine, and quetiapine ; , principally in the patients with OCD who have comorbid tic disorders.174-177 Intravenous clomipramine has also been shown to be more effective than oral administration.178, 179 Two controlled studies were performed to test the.
Nitro reduction Side effects: drowsiness, dizziness, ataxia, dependence, CNS depression, disorientation, psychomotor impairment, confusion, aggression, excitement, falls & vehicle accidents in elderly & anterograde amnesia. Tolerance to sedative hypnotic, muscle relaxant & anticonvulsant, but less tolerance for the anxiolytic & antipanic effects. No cross-tolerance with buspirone & SSRI'S; as well often lacks cross-tolerance with alprazolam. Benzodiazepine withdrawal: Depends on: duration of therapy, dose, rate of tapering & the BZ t life; Onset: 1-2d with short t; 3-8d with long t BZ's; S Sx: insomnia, nausea vomiting, twitching, irritability, anxiety, paresthesias, tinnitus, delirium & even seizures. When D C BZ: If Tx 12 weeks taper at a rate of + -25% week & consider changing to equivalent dose of clonazepam diazepam except alprazolam ; & then taper off the BZ. Length of therapy: Anxiety: use as an adjunct only & re-evaluate q4-6 weeks; Hypnotic: not to exceed 4 week. Caution: BZ & clozapine may lead to marked sedation, salivation, & rare respiratory arrest. Drug interactions DIs ; : CNS depression: antidepressants, antihistamines, barbiturates, ethanol; Antacids absorption; 'd BZ levels by: allopurinol, oral contraceptives, cimetidine, estrogen, erythromycin, fluoxetine, isoniazid, omeprazole, valproic less DI effect on lorazepam, oxazepam, temazepam, but effect on 2-Keto BZ BZ may levels of: digoxin &phenytoin. BZ levels 'd by: carbamazepine, phenobarbital, rifampin & smoking. # Oxidation is a high-energy metabolic pathway impaired in liver disease & reduced in elderly; whereas conjugation to more water soluble glucuronide derivative allows for excretion less intensive ; , thus less affected by DIs. Overdose: safe when taken alone; Treat: flumazenil. Precautions: hx of substance abuse, sleep apnea, cognitive disorder, renal hepatic dx, elderly, porphyria, CNS depression, myasthenia & pregnancy possible teratogen & can precipitate withdrawal in newborns if used in 3rd trimester ; . BZ benzodiazepine DI drug interaction Dx disease * t average range ; can be in geriatric patients & altered by drug interactions Non-formulary in Sask and
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Paperwork filed with the secretary of state says the group formally established itself in late april.
Emiliano zapata site 10-11-04, # 18 jayyy sg enthusiast join date: may 2000 location: fukuoka, japan 3, 145 quote: originally posted by rivas actually clonazepam will space you out its used for people with maniere disease during vertigo attack you can use it with valium but it will just make you more sleepy ativan is used for ppl with maniere disease as well but the magic in ativan is, you can take it under your tongue and it will dissapear in few seconds.
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