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Espite the fact that the back is beautifully engineered and quite strong, lumbar and sciatic pains are among the symptoms for which patients most commonly seek medical advice. in a normal course life, after 24-2 years, dehydration and degenerative changes lead to a loss of height of a lumbar intervertebral disc. the excessive effort, unappropriate positions, the obesity, frequent and long time microtraumatism of the spine truck drivers, gymnasts ; , tall persons increase the propensity for disc rupture. the degenerative changes lead to next stages; disc space narrowing, vacuum fenomenon, disc bulging and disc herniation. The symptoms are low back pain, sciatica unilateral or "in balance" ; , paresthesia, weakness in the affected roots territory. Experienced neurologic consultation by specific manoeuvres Lassegue, osteo-tendinous reflexes control ; can releave the sensorimotor deficits. next step are the imaging investigations. magnetic resonance imaging MRI ; is the mainstay of diagnostic and computed tomographic CT ; myelography is the second choice. Discography, radiography and electromyography may be complementary. after best treated by conservative programs of exercise, courses of physiotherapy, encouragement to pursue their normal activities and the tincture of time, 10-15 % require discectomy. Intractable back pain with sciatica unilateral or "in balance" ; paresthesya, severe foot drop, osteotendinous areflexya or, in emergency, cauda equina syndrome are indications for surgery. the operative techniques like percutaneous discectomy, microsurgical disc surgery, minimal invasive interlamar approach, hemilaminectomy, laminectomy with unilateral or bilateral foraminotomy ; depends by the features of hernia. A clear diagnosis, short period between diagnosis and surgery, surgical team experience and postoperative kynetotherapy, lead to good results. n and lisinopril.
Shoulders, elbows, waist, hips, knees and ankles. "All those exercises are touching on almost every joint, " said Beals. "We might bring a knee up in front of us and extend the leg out then put the foot back down on the bottom of pool. It's a gentle, flowing series of movements sort of like Tai Chi." Pace yourself Pain never should be part of your water workout routine. "Pain is an indicator of doing too much, even in water, " Beals said. "If they find that it hurts them, that's a sign to back off and only do what they feel they can do -- not try to do what their neighbor is doing." Arthritic water workouts last about 45 minutes, but participants are encouraged to set their own pace. They can exercise for as little as 30 minutes or as much as an hour, depending on their individual condition. "And then toward the end, we stretch like you would in land aerobics with the leg behind you, pressing the heel down to the ground, and stretching your calves and your hips, " Beals said. "And then we may hug ourselves to stretch the upper body and then reach the arms down behind us to open the chest up." Facility certification In order to offer a certified aquatic program for arthritis sufferers, facilities must meet certain criteria set forth by the Arthritis Foundation. "For example, it has to have handicap-accessible ; parking no farther than 200 feet from the building, " said Courtney Johnson, public health program coordinator for the Tennessee Arthritis Foundation. "The pool depth must be at least 4 feet, and the air temperature has to be within 5 degrees of the water temperature. A lot of the facilities will have thermometers that floatsin the water at all times. They regularly report their water temperature to the Arthritis Foundation." Approved facilities also must have barrier-free restrooms, walker areas with doorways wide enough for wheelchairs and a locker room area adjacent to the pool. The Arthritis Foundation requires two instructors in the pool per class, or one certified instructor with a lifeguard on duty. "Participants in the classes are not required to be able to swim, due to the shallowness of the water and the presence of two professionals who can help if there is a problem, " Johnson said. A representative from the nearest Arthritis Foundation office will conduct regularly scheduled audits to check that all criteria are being met. Facilities have to submit written agreements that they will provide their programs according to Arthritis Foundation guidelines and submit proof of liability insurance every year.
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Kugiyama K, Yasue H, Okumura K et al: Suppression of exercise-induced angina by magnesium sulfate in patients with variant angina. J Coll Cardiol 1988; 12: 1177-1183. Kuppurajan K, Rajagopalan SS, Rao TK et al: Effect of guggulu commiphora mukul-engl. ; on serum lipids in obese subjects. J Res Indian Med 1973; 8 4 ; : 1-8. Kuppurajan K, Rajagopalan SS, Rao TK et al: Effect of guggulu commiphora mukul-Engl. ; on serum lipids in obese, hypercholesterolemic and hyperlipemic cases. J Assoc Physicians India 1978; 26 5 ; : 367-373. Kushi LH, Folsom AR, Prineas RJ et al: Dietary antioxidant vitamins and death from coronary heart disease in postmenopausal women. N Engl J Med 1996; 334 18 ; : 1156-1162. Kynczl-Leisure M & Cibils LA: Increased bleeding time after magnesium sulfate infusion. J Obstet Gynecol 1996; 175: 1293-1294. Lamm CI, Norton KI, Murphy RJC et al: Congenital rickets associated with magnesium sulfate infusion for tocolysis. J Pediatr 1988; 113: 1078-1082. Lampertico M & Comis S: Italian multicenter study on the efficacy and safety of coenzyme Q10 as adjuvant therapy in heart failure. Clin Invest 1993; 71: 129-133. Langsjoen PH, Langsjoen PH & Folkers K: Long-term efficacy and safety of coenzyme Q10 therapy for idiopathic dilated cardiomyopathy. J Cardiol 1990a; 65: 521-523. Laplaud PM, Lelubre A & Chapman MJ: Antioxidant action of Vaccinium myrtillus extract on human low density lipoproteins in vitro: initial observations. Fundam Clin Pharmacol 1997; 11 1 ; : 35-40. Laurora G, Cesarone MR, De Sanctis MT et al: Delayed arteriosclerosis progression in high risk subjects treated with mesoglycan. Evaluation of intima-media thickness. J Cardiovasc Surg Torino ; 1993 Aug; 34 4 ; : 313-8. Lawson LD: Garlic: a review of its medicinal effects and indicated active compounds, in Lawson LD & Bauer R eds ; : Phytomedicines of Europe: Their Chemistry and Biological Activity. American Chemical Society, Washington, DC, 1998: 176-209. Lee TY & Lam TH: Contact dermatitis due to topical treatment with garlic in Hong Kong. Contact Dermatitis 1991; 24 3 ; : 193-196. Levav AL, Chan L & Wapner RJ: Long-term magnesium sulfate tocolysis and maternal osteoporosis in a triplet pregnancy: a case report. J Perinatol 1998; 15: 43-46. L'Hommedieu CS, Nicholas D, Armes DA et al: Potentiation of magnesium sulfate induced neruomuscular weakness by gentamicin, tobramycin, and amikacin. J Pediatr 1983; 102: 629-631. Li X & Steiner M: Dose response of dietary fish oil supplementations on platelet adhesions and
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Been previously described among P. aeruginosa strains isolated in ICUs [7, 11, 12, 29, but the incidence of cross-acquisition and colonization in the epidemiology of P. aeruginosa in ICUs seems to be variable between hospitals. We found high frequencies of in vitro virulence phenotypes adherence and cytotoxicity ; among both group 1 and group 2 isolates, and the incidence of virulence properties was not statistically different between the two groups. Moreover, there was a strong and statistically significant correlation between adherence and cytotoxicity among the totality of the isolates, indicating that expressing both virulence properties may be essential for P. aeruginosa during infections, and that surveillance isolates may have the same infectious potential as pneumonia-causing isolates. Rajan et al. [31] recently demonstrated that only fully virulent P. aeruginosa capable of coordinately expressing both adhesins and cytotoxins were able to induce apoptosis in respiratory epithelial cells. Moreover, adhesion and expression of the type III secretion system has also been shown to be necessary for efficient apoptosis of Chang epithelial cells by P. aeruginosa [32]. Hostacka and Majtan [33] previously showed high incidence 68.8% ; of bacterial culture filtrate toxicity in a rabbit model vascular permeability activity assay correlated to Vero cell cytotoxicity among P. aeruginosa strains isolated from urine samples of patients after kidney transplantation. In a different study, Bartkova and Ciznar [34] showed that 89% of P. aeruginosa strains obtained from different origins stool, urine, wounds, otitis, sputum, and sewage water ; adhered to undifferentiated epithelial-like HeLa cells. They described the localized and diffuse adherence patterns and showed that Hela cells were a more suitable model for the differentiation of P. aeruginosa strains according to the mentioned types of adherence than CHO and Vero cells. Our results give evidence that the A549 pneumocyte cell-line is also a suitable model to determine the adhesive pattern of P. aeruginosa strains. Moreover, we were able to differentiate another adhesion pattern, termed aggregative. This aggregative pattern was different from the localized one, since it was characterized by the formation of adherent microcolonies after 60 min of bacterial interaction with pneumocyte cells and by the growth of these adherent microcolonies during the interaction time, growth that was absent for the localized adhering strains data not shown ; . These adhesion patterns have been defined by analogy with diarrheagenic Escherichia coli and nosocomial Klebsiella pneumoniae strains [35, 36]. Unlike K. pneumoniae strains, which mainly expressed the aggregative pattern, the P. aeruginosa respiratory tract isolates expressed mainly diffuse adhesion. In K. pneumoniae strains, the localized adhesion phenotype has been associated with multidrug-resistant isolates producing the CAZ-5 SHV-4 -lactamase, but we found no association between the adhesion patterns and antibiotic resistance phenotypes among the P. aeruginosa strains tested. Independently from the adherence patterns, the lowest incidence of virulence properties was encountered among P. aerugi.
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PROPONENT: San Diego County Bar Association STATEMENT OF REASONS: Existing Law: Provides for a one-year lien against a judgment debtor's personal property upon service of an order to appear for judgment debtor's examination Code Civil Proc., 708.110 ; . Neither state law nor bankruptcy law deals specifically with whether this one-year period continues to run during the pendency of the judgment debtor's bankruptcy; although Bankruptcy Code section 108, subdivision c ; , by implication, may be argued to provide that the lien might be extended a maximum of 30 days after notice of the termination of an applicable stay is received by the creditor. This Resolution: Would fill a void in the law, provide a fair mechanism to prevent a judgment debtor from shortening the period of a section 708.110 lien by filing for protection in bankruptcy; and save the judgment creditor from having to resort to expedited hearing procedures just in order to preserve its lien following the debtor's discharge or dismissal from a bankruptcy. The Problem: No law deals with whether a section 708.110 one-year lien continues to expire during the pendency of a judgment debtor's bankruptcy. No law specifically addresses even a 30-day extension of a section 708.110 lien following the termination of a bankruptcy stay. Reactivating a state court case, setting a hearing and convincing a state court judge that such extension is provided under bankruptcy law, all within 30 days, is virtually impossible. Even then, all the judgment creditor can ask the judge to do is extend the lien for some period of time, most equitably for the amount of time that expired while the debtor was protected by the Bankruptcy Code's automatic stay. IMPACT STATEMENT: This resolution does not affect any other law, statute or rule. AUTHOR AND OR PERMANENT CONTACT: Virginia H. Gaburo, Esq., Virginia H. Gaburo & Associates, 4370 La Jolla Village Drive, Suite 980, San Diego, CA 92122; voice 858 ; 546-0183; e-mail vgaburo gaburolaw . RESPONSIBLE FLOOR DELEGATE: Virginia H. Gaburo, Esq.
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