Duty. If another physician is covering for the individual that wrote the order, they are only contacted if the order is ambiguous; it is not processed until clarified. Prescribers are contacted by pharmacy staff by means of calling the individual, sending a text-page which specifies the error, or highlighting the error and replacing the order in the patient chart for the prescriber to review. CONCLUSION The hard stop process was a successful component in decreasing the number of unacceptable orders but requires ongoing vigilance to be continuously successful. Having physician and nurse champions decreases the perception that the pharmacy is a barrier to patient care, and refocuses the issue to patient safety. Text-paging the prescriber that wrote the order is a successful method for notifying them and requires little time for the staff involved. ACKNOWLEDGEMENTS The author wishes to acknowledge the information and support provided by the following individuals: Kate Farthing, PharmD, BCPS Brad S. Fujisaki, RPh Kristine B. Marcus, RPh, BCPS Maureen L. Ober, PharmD REFERENCES.
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Eleni Karabatsaki, Panagiotis Margos, Eleni Kalogeropoulou, Paraskevi Tsiodra, Aristidis Efstratopoulos PURPOSE: It is known that there are various predisposing factors for the presentation of a CVA e.g. hypertension, diabetes ; . Our aim was to appreciate whether there is a relation between the levels of serum uric acid and the presentation of celebrovascular accidents. METHODS: This retrospective study included 320 patients, 154 men 48.12% ; and 166 women 51.88% ; , of mean age 76.8 9.2 years, that have been admitted in our department for a CVA. The patients have been classified according to their clinical findings and their CT findings, in ischaemic strokes, haemorrhagic strokes and TIAs. At least two measurements of serum uric acid have been recorded for each patient. A control group with similar characteristics of sex, age, medication and predisposing factors for CVA, has been included in the study 300 patients, 150 men, 150 women, mean age 77.6 8.8 years ; . RESULTS: From the total of 320 CVAs, 242 were ischaemic 75.6%, Group 1 ; , 38 were haemorrhagic 11.9%, Group 2 ; and 40 were TIAs 12.5%, Group 3 ; . The mean level of serum uric acid in the group of ischaemic CVAs was 5.36 2.4 mg dl, in the haemorrhagic ones was 4.5 1.7 mg dl and in the TIAs was 5.1 1.9 mg dl. The mean level of serum uric acid in the control group Group 4 ; was 5.31 2.14 mg dl. ANOVA has been used to compare the mean values of serum uric acid between the different CVA groups and the control group. The result of the analysis indicated that there is no statistically significant difference in the value of the uric acid between the four groups included in the study p 0.284 ; . CONCLUSION: The above analysis shows that in the existing population there was no statistically significant difference in the levels of uric acid between either the different CVA groups or between the control group and each different CVA group, therefore suggesting that serum uric acid is not a predisposing factor for the presentation of CVAs.
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Box. Patterns of Cross-Reactions Between Acetylsalicylic Acid and Various Nonsteroidal Anti-inflammatory Drugs Common nonsteroidal anti-inflammatory drugs NSAIDs ; that preferentially inhibit cyclooxygenose 1 COX-1 ; and cross-react with acetylsalicylic acid, inducing respiratory and cutaneous pseudoallergic reactions: Diclofenac Diflunisal Etodolac Fenoprofen calcium Flurbiprofen Ibuprofen Indomethacin Ketoprofen Ketorolac tromethamine Meclofenamate sodium Mefenamic acid Nabumdtone Naproxen Naproxen sodium Oxaprozin Piroxicam Sulindac Tolmetin sodium.
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1. The sixth report of Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. Arch Intern Med 1997; 157: 2413 Heart and stroke statistical update. Dallas: American Heart Association, 1998. 3. The fifth report of the Joint National Committee on Detection, Evaluation, and Treatment of High Blood Pressure JNC V ; . Arch Intern Med 1993; 153: 154 Prevention of stroke by antihypertensive drug treatment in older persons with isolated systolic hyper14 and
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When you are taking paroxetine, it is especially important that your health care professional know if you are taking any of the following: aspirin or nonsteroidal anti-inflammatory drugs nsaids ; celecoxib , diclofenac , diflunisal , etodolac , fenoprofen , flurbiprofen , ibuprofen , indomethacin , ketoprofen , ketorolac , meclofenamate , mefenamic acid , meloxicam , nabumetone , naproxen , oxaprozin , phenylbutazone, piroxicam , rofecoxib , sulindac , tolmetin , valdecoxib ; — taking any of these medicines with paroxetine may cause bleeding problems.
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83 American military recruits who later developed RA were compared with matched controls: 60% were rheumatoid factor positive, 62% were anti-CCP positive, 72% were positive for one of either, and 49% positive for both. The antibodies appeared about 4 5 years before diagnosis. The lag time was related to age. It was longer in patients above 40 years of age 22 ; . A French study-group found that the presence of anti-CCP was predictive for RA in both healthy subjects and patients with undifferentiated arthritis 23 ; . In the Leiden early arthritis clinic 93% of patients with unclassified arthritis who were anti-CCP positive at inclusion developed RA within 3 years compared to only 25% of the anti-CCP negative patients 24 ; . In American study on patients with palindromic arthritis, that is an episodic arthritis affecting single but migration joint with the individual attack lasting only a few days, 29 of 61 patients had progressed to RA within 5 years; 83% of these compared to 29% of the patients who remained unclassified had anti-CCP antibodies in their baseline sera 25 ; . A Norwegian 10 year follow-up study showed that RA patients with high levels of anti-CCP were 10 times more likely to develop progression, questioning if anti-CCP antibodies enhance tissue injury in RA? 26 ; . Taken all together the finding raises the question if not anti-CCP should be included as an important parameter in the diagnostic and prognostic clarification of unclassified or preclinical RA. The response to joint inflammation also includes degradation, remodulation, and reparation of connective tissue. During these processes metabolites are liberated and removed to the circulation and finally degraded and or eliminated through the liver or the kidneys. The connective tissue component can be tissue specific, and the metabolites might reflect processes in the various compartment of the joint, that is in the bone, cartilage, synovium or tendons. If it could be documented that their concentration in the circulation in a stoichiometric way reflect the turnover of connective tissue in the joint compartments they might as direct markers give us insight in the local processes. The liberation of metabolites might differ if they are part of a tissue response to pathological inflammatory and not physiological processes, leading to liberation of metabolites not normally found. So far no connective tissue metabolites have proved to be of diagnostic value in RA. However our group has found that the circulating concentration of a number of metabolites is related to disease activity and increased values have been shown in patients with subsequent radiological progression.
Serves as a source of information and education for consumers and providers; promotes family-centered, community-based care that incorporates the needs, perspectives and active participation of affected populations in health programs and policies; and, supports coordinated systems of care by building partnerships, such as among ryan white care act grantees, family planning and prenatal care programs, state aids offices, local health agencies, and other entities and
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Marcos Sanchez-Hanke1, Sabine Kief2, Rudolf Leuwer1, Ulrich Koch1, Ingrid Moll2, Johanna M. Brandner2 ENT-Department, University of Hamburg Medical School, Martinistr. 52, Hamburg, Germany, 2Dermatology, University of Hamburg Medical School, Martinistr. 52, Hamburg, Germany, for example, nabumetone 750mg.
Primary end points analysis from the ATACS trial. Circulation 1994; 89: 81-8. Goodman SG, Langer A. Duries SS, et al. Safety and anticoagulation effect of a low-dose combination of warfarin and aspirin in clinically stable coronary artery disease. Coumadin Aspirin Reinfarction CARS ; Pilot. J Cardiol 1994; 74: 656-7. Hurlen M, Brikssen J, Smith P, et al. Comparison of bleeding complications of warfarin and warfarin plus acetylsalicylic acid: a study in 3166 outpatients. J Intern Med 1994; 236: 299-304. Roth GJ, Calverley DC. Aspirin, platelets, and thrombosis: theory and practice. Blood 1994; 83: 885-98. Kearon C, Hirsh J. Optimal dose for starting and maintaining low-dose aspirin. Arch Intern Med 1993; 153: 700-2. Patrono C. Aspirin as an antiplatelet drug. Drug Therapy 1994; 330: 1287-94. Brooks PM, Day RO. Non-steroidal anti-inflammatory drugs -- differences and similarities. N Engl J Med 1991; 324: 1716-25. Ali M, McDonald JWD. Reversible and irreversible inhibition of platelet cyclooxygenase and serotonin release by nonsteroidal and antiinflammatory drugs. Thromb Res 1978; 13: 1057-65. Amadio P, Cummings DM, Amadio P. Non-steroidal anti-inflammatory drugs. Tailoring therapy to achieve results and avoid toxicity. Postgrad Med 1993; 93: 73-97. Simon LS, Mills. JA. Non-steroidal anti-inflammatory drugs. N Engl J Med 1980; 302: 1237-43. Kallis P. Tooze JA, Talbot S, et al. Pre-operative aspirin decreases platelet aggregation and increases post-operative blood loss -- a prospective, randomized, placebo controlled, double-blind clinical trial in 100 patients with chronic stable angina. Eur J Cardiothorac Surg 1994; 8: 404-9. Eauno P, Petersen KD, Husted SE. Increased blood loss after preoperative NSAID. Retrospective study of 186 hip arthroplasties. Acta Orthop Scand 1993; 64: 522-4. Robinson CM, Christie J, Malcolm-Smith N. Non-steroidal anti-inflammatory drugs, perioperative blood loss, and transfusion requirements in elective hip arthroplasty. J Arthroplasty 1993; 8: 607-10. Kenny GNC. Potential renal, haematological and allergic adverse effects associated with nonsteroidal anti-inflammatory drugs. Drugs 1992; 44: 31-7. Jacobus RBJ, Brouwers de Sinel P. Pharmacokinetic-pharmacodynamic drug interactions with nonsteroidal anti-inflammatory drugs. Clin Pharmacokinet 1994; 27: 462-85. Wells PS, Holbrook AM, Crowther NR, et al. Interactions of warfarin with drugs and food. Ann Intern Med 1994; 121: 676-83. O'Callaghan JW, Thompson RN, Russell AS. Combining NSAIDs with anticoagulants: yes and no. Can Med Assoc J 1984; 131: 857-9. Hilleman DE, Mohiuddin SM, Lucas BD Jr. Nonsteroidal antiinflammatory drug use in patients receiving warfarin: emphasis on nab8metone [review]. J Med 1993; 95 2A ; : 30S-34S and
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Takes the values of 914 i.e., a range around the `tipping point' 0 otherwise.4 In a similar fashion, we create dummies for the ranges of 0 sources; 14 sources; 58 sources; and for 916 sources. The results of this exercise are reported in Table 5, Model IV for reasons of space, the results are reported for INNWORLD only ; . The results show that the dummies below the benchmark all have a negative sign--as expected--but only the dummy for the value of 0 is significant. The dummy above the benchmark is positive but insignificant. Further experimentation with dummies reveals that if a dummy for the value of 16 sources is created separately not shown for reasons of space ; , such a dummy gets a negative sign, but it is not significantly different from the benchmark at a conventional level of significance. The results indicate that while there are decreasing returns in innovation for increased openness in terms of breadth, no negative returns are to be detected. We find rather strong support for the hypothesis that external search depth--the extent to which firms draw intensively from different sources of innovative ideas--is curvilinearly taking an inverted U-shape ; related to innovative performance Hypothesis 2 ; , since the variable measuring the DEPTH of openness and the squared term are both significant and have the expected, because nabumetpne abuse.
Labs unsure whether to join stem cell bank When President Bush promised to create a stem cell bank for the federally funded 22 strains of stem cell lines, he thought he was doing a commendable job. However, there has been a backlash of criticism from those research facilities who hold some of the strains. They are hesitant to give them up without some strict guidelines as to preservation, storage, and distribution. Other research facilities see the government's stem-cell research as outdated. For instance, the strains used were preserved using mice cells and therefore impossible to use in a human body for further testing. As the medical industry continues to best itself, the 22 strains are becoming more and more worthless to the ever-mutating industry. Yahoo News -- December 6, 2005 : news.yahoo s ap 20051206 ap on he stem cell bank; ylt AoIp3WWBa5P.SUTyJ1c34LGs0NUE; ylu X3oDMTA3czJjNGZoBH NlYwM3NTEA California ruling sends 2 stem cell suits to trial People's Advocate and the National Tax Limitation Foundation as well as the California Family Bioethics Council have filed a lawsuit hoping to block $3 billion in state bonds that would be used to fund their stem cell research initiative. Judge Bonnie Lewman Sabraw declined the dismissal meaning a trial must ensue -- further pushing back the availability of these funds. A year has already passed since the California natives voted to create this pool of money and since whichever party loses the trial will most likely appeal, the delay is most likely going to be longer. The request for dismissal arose from the various organizations' claims that the structure would violate California's Constitution by spending uncontrolled money. Judge Sabraw's response was that the organization had not met their claim of proving the stem cell initiative to be "clearly, positively and unmistakably unconstitutional." New York Times -- November 30, 2005 : nytimes 2005 12 01 business 01stem ? ei 5070&en a5eab208b5345785&ex 1136955600&pagewanted print and
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Table 2: Alcohol and drug use by participants Substance s ; found Alcohol only 2 Alcohol and controlled drugs Alcohol and prescribed drugs Alcohol, prescribed and controlled drugs Drugs only, prescribed & controlled Total No. 63 41 8 % 52.5 34.2 6.7 Of those participants for whom information was available, almost half admitted to have willingly taken recreational drugs not including alcohol ; prior to the assault see Table 3 below for details ; . Over half of all participants 65, 54% ; were found to have drugs in their blood or urine. Twenty-eight 43% ; of those that tested positive for drugs showed evidence of drugs that they had not admitted to in the questionnaire although definitive information on willingly taken drugs was not available in a further 17 cases.
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NOTE: This serves as notification of an amendment to your existing Performance Extra Program criteria as stated in PCP Attachment A of the Primary Care Physician Addendum to the Anthem Services, Inc. Professional Provider and Preferred Professional Provider Agreements, as applicable; and or your HealthKeepers, Inc., Peninsula Health Care, Inc., Priority Health Care, Inc. Primary Care Physician or Center Agreement as indicated in Exhibit G, as applicable. If you object to this modification, you have all the rights available to you described in the amendment paragraph of your Primary Care Physician Addendum or your HealthKeepers, Inc., Peninsula Health Care, Inc., Priority Health Care, Inc. Primary Care Physician or Center Agreement and pletal.
| Nabumetone tabs tabletsMeda AB, the Swedish Specialty Pharmaceutical Company, has agreed to a deal with the GlaxoSmithKline Group GSK ; on products for local treatment of inflammatory diseases in the distal bowel, marketed under the brand names Colifoam and Proctofoam. Meda has also acquired Epifoam, a product development of Proctofoam , which Meda plan to introduce to the markets. Colifoam and Proctofoam are marketed by GSK in many European countries and in a few overseas markets. Meda acquires the products in all the GSK markets. At present, the majority of sales is generated in the UK market. The annual turnover of the products is around 55 MSEK and the agreement is structured as a cash deal where GSK will receive GBP 7, 9m. "We are very satisfied to announce this deal with GSK. The products fit perfectly into one of Meda's major therapy areas, the gastrointestinal. Meda is currently marketing Colifoam in Sweden, and through this deal we acquire products that we already know and like. We can now broaden our territory and increase our gross margin in Sweden. And above all, it's a significant contribution to our UK operations. We are also very glad to have finalised another product acquisition from the GSK group. One year ago Meda acquired Relifex nabumetone ; from GSK", says Anders Lnner, CEO of Meda. "Meda has proven itself to be a good partner for Colifoam in Sweden. We are pleased to have concluded this agreement and we are confident that the products are in good hands", says John Clarke, President, GlaxoSmithKline Consumer Healthcare, Europe.
Corresponding: Dr George K. K. Lau, University Department of Medicine, Queen Mary Hospital, 102 Pokfulum Road, Hong Kong, SAR, China. Email: gkklau netvigator Grant support: This project was supported with a grant from the Cheng Si-yuan China-International ; Hepatitis Research Foundation to the University of Hong Kong ; , China National 973 research grant G 1999 054105 to GKK Lau.
A 7-year-old girl went to an urgent care centre with symptoms of an ear infection. The doctor prescribed 15 mg milligrams ; of codeine syrup every 4 hours as needed for 7 days. The pharmacist prepared the bottle of syrup with a label that read "give 15 mL millilitres ; every 4 hours as needed." The mother left the pharmacy with a bottle containing 600 mL of codeine syrup. The mother thought 600 mL seemed like a lot of syrup compared to other prescriptions she had received of about 250 mL so she looked at the information on the label. From the information, she calculated that giving 15 mL equalled a 75 mg dose, which was significantly more than the 15 mg the doctor had ordered. Fortunately, she returned to the pharmacy and asked the pharmacist to double check the original prescription. The pharmacist realized that mL volume of liquid ; was used instead of mg dose of medication ; . The directions on the bottle should have read "give 3 mL every 4 hours if needed". This would then equal the dosage on the prescription the doctor had originally written. The label was retyped and the mother went home with the correct amount of codeine syrup. There could have been a serious overdose problem if the mother had not caught the error before she started giving her child the medication. If something seems unusual when you receive your prescription, don't hesitate to ask the pharmacist to double check the original prescription.
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Typically, the delivered condensation aerosol results in a peak plasma concentration of indomethacin, ketoprofen, celcoxib, rofecoxib, meclofenamic acid, fenoprofen, diflunisal, tolfenamic acid, naproxen, ibuprofen, flurbiprofen, or nabumetone in the mammal in less than 1 preferably, the peak plasma concentration is reached in less than 5 more preferably, the peak plasma concentration is reached in less than 2, 1, 05, or 005 h arterial measurement.
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Bradley Pharmaceuticals, Inc. and Subsidiaries NOTES TO CONSOLIDATED FINANCIAL STATEMENTS December 31, 2002 All warrants listed above are exercisable. The number of shares covered by each outstanding warrant, and the exercise price, must be proportionately adjusted for any increase or decrease in the number of issued shares resulting from a subdivision or consolidation of shares, stock split, or the payment of a stock dividend, and are summarized as follows.
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Appeal from the Order Entered May 18, 2000, In the Court of Common Pleas of Lehigh County Civil Division, No. 97-C-1535 ALLEN TRACH Appellant v. J. FELLIN AND THRIFT DRUG ECKERD DRUG STORE, THRIFT DRUG, INC. AND ECKERD DRUG CO. Appellee : : : THE SUPERIOR COURT OF PENNSYLVANIA.
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