Pheniramine
Aluminum hydroxide Aluminum hydroxide magnesium hydroxide Brompheniramine phenylephrine elixir Calcium glubionate 1.8 g 5 mL Calcionate ; Chlorpromazine 100 mg mL Ferrous sulfate 220 mg 5 mL Fluoroquinolones: ciprofloxacin Cipro ; , gatifloxacin Tequin ; , levofloxacin Levaquin ; ofloxacin Floxin ; Guaifenesin 20 mg mL Robitussin ; Lithium citrate 8 mEq 5 mL * Medium chain triglyceride oil MCT Oil ; Metoclopramide 1 mg mL Roxane ; Opium tincture, camphorated, 0.04% elixir Paregoric ; Potassium chloride 10% liquid Potassium chloride 20% liquid Pseudoephedrine 6 mg mL Rugby ; Sodium biphosphate 480 mg mL Fleets Phospho-soda ; Sucralfate Carafate ; Thioridazine 30 mg mL; 100 mg mL Zinc sulfate capsules.
Pheniramine dosageInformation on before taking this medication, tell your doctor if you are taking any of the following medicines: antihistamines such as brompheniramine dimetane, bromfed, others ; , chlorpheniramine chlor-trimeton, teldrin, others ; , azatadine optimine ; , clemastine tavist ; , and many others; narcotics pain killers ; such as meperidine demerol ; , morphine ms contin, msir, others ; , propoxyphene darvon, darvocet ; , hydrocodone lorcet, vicodin ; , oxycodone percocet, percodan ; , fentanyl duragesic ; , and codeine fiorinal, fioricet, tylenol #3, others sedatives such as phenobarbital solfoton, luminal ; , amobarbital amytal ; , and secobarbital seconal phenothiazines such as chlorpromazine thorazine ; , fluphenazine prolixin ; , mesoridazine serentil ; , perphenazine trilafon ; , prochlorperazine compazine ; , thioridazine mellaril ; , and trifluoperazine stelazine or antidepressants such as doxepin sinequan ; , imipramine tofranil ; , nortriptyline pamelor ; , fluoxetine prozac ; , paroxetine paxil ; , sertraline zoloft ; , phenelzine nardil ; , and tranylcypromine parnate! RESEARCH PAPERS AND REFEREED ARTICLES Fourie, M. 2000. A writer's story: A narrative analysis of human occupation. The South African Journal of Occupational Therapy 30 2 ; : 3-7. CONFERENCE ABSTRACTS Dawe, A. L. & Weskamp, K. 2000. The effect of biomechanical and activity analysis on the management of people with arthritis. Presented at the 6th Congress of the European Federation of Societies for Hand Therapy, Barcelona, Spain. De Jager, L. T. & Weskamp, K. 2000. Tendon transfer in a 3 year old child with tetraplegia. Poster presented at the 7th Congress of the European Societies for Surgery of the Hand, Barcelona, Spain. De Jager, L. T. & Weskamp, K. 2000. Tendon transfers in the hand of a child with tetraplegia. Presented at the 31st Congress of the South African Society for Surgery of the Hand, Durban, South Africa. Duncan, M.E. 2000. Vocational rehabilitation in forensic psychiatry: A tertiary hospital perspective. Presented at the Western Cape Psychosocial Rehabilitation Conference: Crossing Boundaries Towards Quality in Mental Health Care, Cape Town, South Africa. Furneaux, M., Percy, S., Roberts, C. & Seider, L. 2000. The influences on the experience of work of employees with mental illness in the open labour market: A collective case study. Presented at the South African Federation of Mental Health 2000 Conference: Towards Equal Employment for People with Mental Illness and Mental Handicap, Johannesburg, South Africa. Isaacs, R. 2000. Exit: Come and talk about your stay. Poster presented at the Western Cape Psychosocial Rehabilitation Conference: Crossing Boundaries Towards Quality in Mental Health Care, Cape Town, South Africa. Lorenzo, T., Mdlokolo, P., January, M. & Mguda, N. 2000. The Value of Ubuntu in social and economic transformation: A renewed hope through storytelling for disabled women in South Africa. Poster presented at two Conferences of Uppsala University, one on Disability and the other on Gender, Power and Culture, Uppsala, Sweden. Van Niekerk, L. & Lorenzo, T. 2000. Establishing indicators of outcome through the process of participatory action research. Presented at the First Qualitative Evidence-based practice conference, Coventry, United Kingdom. Watson, R.M. 2000. Learning how to help learners learn. Presented at the 6th Annual International Qualitative Health Research Conference, Banff, Canada. Weskamp, K. & Dawe, A. L. 2000. The importance of biomechanical and activity analysis in the treatment of arthritis: A case study. Presented at the 31st Congress of the South African Society for Surgery of the Hand, Durban, South Africa. 124, for example, chlortrimeton. Personalize everyday health center for helping me. Name of drug Paracetamol TM ; Chloroquine phosphate INN ; Cotrimoxazole INN ; Amoxicilline INN ; Acetyl salicytic acid INN ; Mebendazole INN ; Quinimax TM ; Ibuprofene INN ; N-butyl-hyoscine INN ; Quinine dihydrocloride INN ; Metronidazole INN ; Benzylpeniciline INN ; Dexachlorpheniramine INN ; Tetracycline ophy. ointm. INN ; Metoclopramide INN ; Diazepam INN ; Ampicilline INN ; Aspirine TM ; Buscopan TM ; Fansidar TM ; other 0% 5% 10% 15% before n 1387 ; during n 752 ; after n 680 and progesterone. Gastroesophageal reflux GER ; or GI dysmotility: Infants and children: 0.10.2 mg kg dose up to QID IV IM PO; max. dose: 0.8 mg kg 24 hr Adult: 1015 mg dose QAC and QHS IV IM PO Antiemetic: 12 mg kg dose Q26 hr IV IM PO. Premedicate with diphenydramine to reduce EPS. For aafa, she will be answering the telephones, putting together informational packets, assisting in health fairs and informational-type exhibits and propafenone, for example, brompheniramine maleate! A quantitative study of the action of synthetic oxytocin on the pregnant human uterus. J Pharmacol Exp Ther 121: 18-25, 1957. Takahashi K, Diamond F, Bieniarz J, et al: Uterine contractility and oxytocin sensitivity in preterm, term and post-term pregnancy. JObstetGynecol 136: 774-779, 1980. Pheniramine maleate naphazoline hydrochlorideFunction. First-generation H1 antihistamines are still used in the treatment of urticaria despite the low concentrations achieved in skin129, 130 and their poor benefit-to-risk ratio. In time-honored tradition, they are often given three or four times daily; however, this regimen is unnecessary, because most of them e.g., hydroxyzine, chlorpheniramine ; have elimination half-life values of approximately 24 hours see Table 51-3 ; . Combinations of first-generation H1 antihistamines such as hydroxyzine and cyproheptadine, or of a first- and a secondgeneration H1 antihistamine such as cetirizine in the morning and hydroxyzine at night ; , are recommended empirically by some highly respected physicians.216 These regimens have not been tested in prospective, randomized, placebo-controlled, double-blind trials; moreover, the administration of a firstgeneration H1 antihistamine at night places patients at risk for an antihistamine "hangover" sedation with or without psychomotor impairment ; the next morning.11 Sequential use of two different second-generation H1 antihistamines in the same day is also recommended on an empiric basis.216 In urticaria, as in allergic rhinoconjunctivitis, the relatively flat dose-response curve for H1 antihistamine efficacy see Figure 51-12 ; contrasts with the steep dose-response curve for adverse effects, particularly for first-generation H1 antihistamines. In patients with acute urticaria, chronic urticaria, or dermographism, combined treatment with an H1 and an H2 antihistamine may be more effective in reducing symptoms than treatment with an H1 antihistamine alone. The main contribution of H2 antihistamines is to down-regulate vascular and pyrazinamide. Pheniramine pregnancyDRUG NAME C cefpodoxime cefprozil CEFTIN suspension ceftriaxone cefuroxime CELEBREX CELLCEPT CELONTIN cephalexin CEREBYX CEREZYME cerovel cesia CHEMET chloral hydrate chloramphenicol CHLORHEXIDINE chlorhexidine gluconate chloroquine chlorothiazide chlorpheniramine chlorpromazine chlorpropamide chlorthalidone chlorzoxazone cholestyramine, light choline mag trisal ciclopirox cilostazol cimetidine CIPRO IV in D5W CIPRODEX ciprofloxacin INJ injectable form only QLL quantities may be limited 2 Antiinfectives Antiinfectives Antiinfectives Antiinfectives Antiinfectives Musculoskeletal Medications Antineoplastic Immunosuppressant Drugs Autonomic & CNS Medications Antiinfectives Autonomic & CNS Medications Endocrine Medications Dermatological Medications OB & GYN Medications Diagnostic & Misc. Medications Autonomic & CNS Medications Antiinfectives Antiinfectives Ear-Nose-Throat Medications Antiinfectives Cardiovascular Medications Respiratory Medications Autonomic & CNS Medications Endocrine Medications Cardiovascular Medications Musculoskeletal Medications Cardiovascular Medications Musculoskeletal Medications Antiinfectives Nutrition, Blood Modifiers, Electrolytes Gastrointestinal Medications Antiinfectives Ear-Nose-Throat Medications Antiinfectives PAR authorization may apply ST step therapy 58 TIER NOTES MEDICAL CONDITION and seroquel. Reasons why some isolates are not viable in vitro include selfmedication before consultation that is undetected by a standard urine test; unexplained factors that transform in vitro most of the ring forms into gametocytes, which do not undergo nuclear division and thus do not incorporate a significant amount of radiolabeled DNA precursor; and serum factors that inhibit parasite growth. Thirdly, the rapid in vitro test is probably not sensitive enough to detect the difference in chloroquine incorporation in chloroquine-resistant isolates when the parasitemia is below 1%. Chloroquine is known to accumulate in uninfected erythrocytes, leading to diminished sensitivity of the rapid in vitro test at low parasitemias 22 ; . Initial experiments were done with culture-adapted parasites at 1% parasitemia 11 ; . The data obtained by Gluzman et al. do not display distinct differences between the chloroquine-sensitive and the chloroquine-resistant P. falciparum strains below 1% parasitemia. Since a large majority of malaria-infected patients present with a parasitemia of 1%, the rapid test may not be suitable for clinical isolates. In addition, the threshold values for chloroquine resistance based on laboratory-adapted P. falciparum clones may not be applicable to the parasite isolates. The above-mentioned technical considerations, as well as the poor performance of the rapid in vitro test compared with the isotopic in vitro tests and the in vivo test, preclude any practical application of the rapid in vitro test in clinical isolates without further technical improvements. However, the rapid in vitro test may be considerably improved and may prove to be an accurate diagnostic tool for chloroquine resistance if the incubation period is extended to 12 to even to 42 h. Previous in vitro studies have shown that chloroquine-sensitive and chloroquine-resistant fresh isolates of P. falciparum can be distinguished by the property of resistance modulators e.g., verapamil, amlodipine, desipramine, cyproheptadine, chlorpheniramine, and chlorpromazine ; to decrease the level of resistance in chloroquine-resistant parasites but not in chloroquine-sensitive isolates 14 ; . These studies were designed to determine the IC50s, requiring the standard incubation time of 42 to allow parasite maturation to the schizont stage in test plates with a full range of chloroquine concentrations. The obvious drawback to this proposed technical modification is the loss of rapidity in obtaining the results. Concordance between the isotopic in vitro tests and the in vivo test was moderate. Several factors explain why the level of concordance was not higher, as was expected. Parasite clearance in malaria-infected patients depends on various pharmacodynamic and pharmacokinetic parameters, and the level of acquired immunity interacts with and enhances drug efficacy 17 ; . A patient harboring chloroquine-resistant populations of P. falciparum, as determined by an in vitro test, may thus eliminate all parasites after an adequate treatment with chloroquine, due to the "booster effect" of the immune system. A patient infected with chloroquine-sensitive parasites, on the other hand, may fail to clear the parasites within 14 days because of an inadequate plasma chloroquine level or reinfection a few days before or after chloroquine treatment is administered. In the latter case, the new populations of chloroquine-sensitive or chloroquine-resistant parasites may emerge in the peripheral blood circulation when a subtherapeutic plasma chloroquine level has been attained before day 14. It is important to note that the in vivo test measures the proportion of therapeutic failure in a given patient population, which may or may not be directly related to drug resistance, while in vitro tests measure the capacity of parasites to grow under different concentrations of drugs. Isotopic in vitro tests seem to be more objective and more accurate in characterizing the phenotype of. Corresponding Author: Shigeo Yamamura, School of Pharmaceutical Sciences, Toho University, Miyama 2-2-1, Funabashi, Chiba, 274-8510, Japan, yamamura phar.toho-u.ac.jp and quinine. To an inadequate blood examination, false positive results are very common when inexperienced persons examine blood smears for malaria. Pseudoepidemics of malaria have also been caused from over-interpreting normal cellular elements in an effort not to miss a serious treatable disease. Experience in reading blood slides and knowledge of when to obtain a smear are valuable in medical personnel deploying to the tropics. Newer Diagnostic Tools In an effort to eliminate some of the subjectivity from the reading of malaria blood films, several new tests to measure parasites in the blood have been developed. They have not yet replaced blood smears, and any military medical unit deploying to the tropics should have the capacity to examine blood films until more experience is gained with the newer antigenic or nucleic acid detection methods. Microscopic examination can be speeded through the use of fluorescent dye in a capillary tube, but this method still requires the ability to interpret microscopically visible parasites.20 Fixed antigen detection methods using either the histidine-rich protein II or parasite lactate acid dehydrogenase of P falciparum have shown promising results and give an answer that is relatively easy to interpret.21 The usefulness of such techniques in the field remains to be proven. The polymerase chain reaction holds promise as a diagnostic method for a multitude of infectious agents including malaria, but it is still a research tool and is not soon expected to be useful in the field because of the level of technology required for accurate analysis.22 Rapid diagnostic methods are most likely to be useful in situations where large numbers of samples need to be tested quickly. Until the technology greatly improves, microscopic examination of stained blood films remains the best way to determine if any single service member has malaria. Recommendations for Prophylaxis, Therapy, and Control Prophylaxis Chemoprophylaxis can kill or suppress the parasites, but a continuous concentration of drug must be maintained. Prophylactic and treatment regimens recommended vary over time because of evolving resistance patterns and other factors; it is critical that medical officers consult with command medical authorities to ensure regimens are current. Marijuana is the most commonly used illicit drug among teens and rebetol. Health Information Technology is vital to this process. There must be a national commitment to providing financial and technical assistance to America's health care providers in order to facilitate their transition into the digital age. Chlorpheniramine loratadine doxylamine brompheniramine phenindamine pheniramjne triprolidine and diphenhydramineSome companies claim to sell a generic version, but those medications are fake, substandard, and potentially dangerous and requip. Plasma concentrations after single doses used clinically. Derived from distribution of 14C-labeled drug in rats for chlorpheniramine, tripelennamine, hydroxyzine, and promethazine and derived from human autopsy specimens for diphenhydramine. Brompheniramine [1] [2] [3] Total Brompheniramine + phenylephrine [4] [5] Narayan et al. 1982 ; Lange et al. 1983 ; RLND RLND 12 mg day p.o. 24 mg + 30 mg + 30mg day p.o. Chlorpheniramine + phenylpropanalamine [6] [7] [8] [9] [10] [11] [12] Total Dextroamphetamine [13] Ephedrine [14] [15] Gilja et al. 1994 ; Proctor and Howards 1983 ; Kedia et al. 1977 ; 17 Diabetes mellitus, 8 RLND RLND 50 mg day p.o. 100 mg day p.o. 4 weeks 2 h4 days 25 1 4 unclear unclear unclear 2.26 unclear 10 non-responder, same patients as [28] same patients as [13, 17, 39] Unclear 4 30 Proctor and Howards 1983 ; Narayan et al. 1982 ; RLND 10 mg day + 16 mg day p.o. 150 mg day p.o. + 7550 mg day p.o. 75 mg day p.o. 75 mg day p.o. 75 mg day p.o. 30 mg day p.o. 75 mg day p.o. stepwise reduction to maintenance 50 mg day p.o. 2550 mg day p.o. 25 mg day p.o increasing dose from 25150 mg day p.o. 75 mg day p.o. 50 mg day p.o. 25 mg day p.o. for 3 days + 50 mg day p.o. last 4 days 2050 mg day p.o. unclear 2 h4 days 1 6 unclear 2.5 11 Proctor and Howards 1983 ; RLND 20 mg day p.o. 2 h4 days 1 unclear 2 11.5 Stewart and Bergant 1974 ; Thiagarajah et al. 1978 ; Virupannavar and Tomera 1982 ; Kragt and Schellen 1978 ; Sandler 1979 ; Check et al. 1990 ; Zavos and Wilson 1984 ; Urethral stricture Idiopathic Unclear 2 Bladder neck surgery, 1 RLND 50 mg day p.o. 50 mg day p.o. unclear unclear unclear unclear unclear 1 4 3 unclear 0.050.4 0.055 530 unclear 1137 1160 unclear unclear unclear 20 failures to [241] Diabetes mellitus RLND Urethral stricture 50 mg day p.o. 50 mg day p.o. 50 mg day p.o. unclear 12 h 210 days 1 unclear normal 152160 unclear normal 20 unclear 2 weeks 2 weeks 10 7 3 unclear 53 unclear Brompheniramine + phenylephrine + phenylpropylamine Andaloro and Dube 1975 ; Budd 1975 ; Narayan et al. 1982 ; Diabetes mellitus Diabetes mellitus RLND 16 mg day p.o. 16 mg day p.o. 24 mg day p.o. 12 h 3 days 2 weeks 1 6 unclear unclear 2 unclear unclear 50 unclear unclear 60 dry mouth failures from [4]. Text book of organic medicinal and pharmaceutical chemistry. 11st edition. Editor: Jaime N. Delgado and William A. Remers. Lippincott-Raven Publishers, New York, USA, 1998. Foye's principles of medicinal chemistry. 5th edition. Editor: Davide A. William and Thomas L. Lemake. Lippincott Williams & Wilkins, USA, 2002. Introduction to medicinal chemistry, how drugs act and why. Editor: Alex Gringauz. Wiley-VCH, USA, 1996. Basic & clinical pharmacology. Editor: Bertram G. Katzung. The McGraw-Hill Companies, Inc., New York, USA, 2001. Medication Error": The observed preparation or administration of drugs or biologicals which is not in accordance with: 1 ; Physician's orders; 2 ; Manufacturer's specifications not recommendations ; regarding the preparation and administration of the drug or biological; 3 ; Accepted professional standards and principles which apply to professionals providing services. Accepted professional standards and principles include the various practice regulations in each State, and current commonly accepted health standards established by national organizations, boards, and councils. "Significant medication error" means one which causes the resident discomfort or jeopardizes his or her health and safety. Criteria for judging significant medication errors as well as examples are provided under significant and non-significant medication errors. Discomfort may be a subjective or relative term used in different ways depending on the individual situation. Constipation that is unrelieved by an ordered laxative that results in a drug error that is omitted for one day may be slightly uncomfortable or perhaps not uncomfortable at all. When the constipation, for example, robitussin. 6. Loratadine 7. Pheiramine Aminosalicylate 8. Promethazine Hydrochloride and progesterone. When a standard drug solution was assayed repeatedly n 6 ; , the mean error accuracy ; and relative standard deviation precision ; were found to be 6% and 8% respectively. Stood. On the other hand, it has been suggested that the intestinal epithelia function as an absorptive barrier for drug absorption 4, 14, 17 ; . Intestinal secretion of organic cations was first demonstrated in isolated guinea pig intestinal mucosa 30, 31 ; . It was shown that P-glycoprotein localized in the intestinal brushborder membrane is involved in the active intestinal secretion of organic cations 5 ; . A guanidine H exchanger was characterized in intestinal brush-border membrane vesicles from rabbit 20 ; . It has been suggested that the guanidine H exchanger functioned as an efflux system for the organic cations. Furthermore, it was demonstrated that ciprofloxacin, a fluoroquinolone, was secreted by another secretory system distinct from both the guanidine H exchanger and P-glycoprotein 3 ; . However, the details of these intestinal transport systems for organic cations are not well understood at present. Recently, we have demonstrated that diphenhydramine, an antihistamine, is accumulated by the pHdependent transport system in Caco-2 cells 21 ; . Diphenhydramine accumulation was temperature dependent, saturable, and shown to be decreased at lower extracellular pH. Diphenhydramine accumulation was not inhibited by tetraethylammonium or biological amines and or neurotransmitters such as histamine, serotonin, dopamine, and choline. However, cellular accumulation of diphenhydramine was affected by cis- and transinteraction with another structurally similar organic cation, chlorpheniramine. Therefore, it was suggested that a specific transport system for diphenhydramine is present in Caco-2 cells. The purpose of the present study was to elucidate further the substrate specificity and pH dependence of this transport system. Our findings indicate that diphenhydramine transport in Caco-2 cells is mediated by a novel pH-dependent tertiary amine transport system that recognizes Ndimethyl or N-diethyl moieties. Drug Name b-vex carbinoxamine maleate cardec ceron ceron C-HIST SR chlor pseudo sr chlorex-a 12 chlorex-a chlor-mes jr chlorpheniramine maleate er chlorpheniramine maleate tr chlorpheniramine maleate phenylephrine hcl chlorpheniramine tannate phenylephrine tannate chlorpheniramine phenyltoloxamine phenylephrine chlorpheniramine pseudoephedrine sr chlorpheniramine pseudoephedrine clemastine fumarate clemastine fumarate CODIMAL L.A. HALF codimal l.a. coldamine coldex-a sr colfed-a COMHIST cophene #2 c-phed tannate c-phen c-phen cpm 8 pe 20 msc 1.25 cpm 8 pse 90 msc 2.5 cyproheptadine hcl cyproheptadine hcl DALLERGY JR dallergy DALLERGY DALLERGY DALLERGY DALLERGY-JR d-amine-sr DECON-A DECONAMINE SR DECONAMINE DECONAMINE decongestine tr dehistine denaze dexchlorpheniramine maleate cr DEXCHLORPHENIRAMINE MALEATE 95. Lipostat Tab 40mg Simvastatin Tab 10mg Simvastatin Tab 20mg Simvastatin Tab 40mg Simvastatin Tab 80mg Zocor Tab 10mg Zocor Tab 20mg Zocor Tab 40mg Acrivastine Cap 8mg Acrivastine Pseudoephed Cap 8mg 60mg Semprex Cap 8mg Benadryl Allergy Relief Cap 8mg Mizolastine Tab 10mg M R Mizollen Tab 10mg Desloratadine Tab 5mg Neoclarityn Tab 5mg Levocetirizine Tab 5mg Xyzal Tab 5mg Optimine Syr 0.5mg 5ml Loratadine Tab 10mg Loratadine Syr 5mg 5ml Clarityn Tab 10mg Clarityn Syr 5mg 5ml Fexofenadine HCl Tab 120mg Fexofenadine HCl Tab 180mg Telfast 120 Tab 120mg Telfast 180 Tab 180mg Brompheniramine Mal Elix 2mg 5ml Dimotane Tab 4mg Dimotane Elix 2mg 5ml Dimotane L.A. Tab 12mg Chlorphenamine Mal Inj 10mg ml 1ml Amp Chlorphenamine Mal Oral Soln 2mg 5ml Chlorphenamine Mal Tab 4mg Piriton Tab 4mg Piriton Syr 2mg 5ml. Lobes, and the superior temporal regions. In patients with severe AD, the same regions are affected, but the hypometabolism is much more pronounced with sparing only of the sensorimotor, visual, and subcortical areas Fig. 3 ; . Longitudinal studies have shown that CMRGlc values decrease more rapidly over time in patients with AD than age-matched control subjects and particularly affect the temporal, parietal, and frontal lobes [21]. The authors' research group has also developed a semiquantitative subjective scoring system designed to assess for disease severity in AD patients [18]. Such a scoring system, which weighs metabolic values according to the areas particularly involved in AD, such as the parietal, temporal, and frontal lobes, may be beneficial for routine clinical use and future research studies of therapeutic interventions. PET imaging also provides the ability to measure changes in neurotransmitter systems that might be affected in AD. One study demonstrated significant decreases in acetylcholinesterase activity in the neocortex, hippocampus, and amygdala of all patients with AD, suggesting a loss of cholinergic innervation in the basal forebrain [22]. The temporal and parietal cortices were the most affected, although reductions were relatively uniform in the cerebral neocortex. PET can also play an important role in the evaluation of therapeutic interventions for AD. The relatively recent development of several pharmaceuticals for, for example, rondec.
Blenoxane, see Bleomycin sulfate Bleomycin sulfate 15 units Botulinum toxin type A per unit 5Botulinum toxin type B per 100 units Brethine, see Terbutaline sulfate or Terbutaline, compounded Bricanyl Subcutaneous, see Terbutaline sulfate Brompheniramine maleate per 10 mg Bronkephrine, see Ethylnorepinephrine HCl Bronkosol, see Isoetharine HCl Budesonide inhalation solution, concentrated form 0.25 mg Budesonide inhalation solution, unit dose form 0.25 mg Buprenorphine Hydrochloride 0.1 mg Busulfan 2 mg IM, IV, SC IM IM J9040 J0585 J0587.
NEW YORK STATE DEPARTMENT OF HEALTH 07 20 2007 LIST OF MEDICAID REIMBURSABLE DRUGS PRICING ERRORS ARE NOT REIMBURSABLE PRICES EFFECTIVE 07 20 2007 MRA COST -0.53586 0.55857 0.43807 0.46537 -0.25875 0.44962 0.45375 0.44962 -0.24900 0.00507 0.01105 0.00504 -0.00600 0.00600 0.00591 -0.00219 0.00219 COST ALTERNATE -FORMULARY DESCRIPTION 2 MG TABLET DEXAMETHASONE 2 MG TABLET DEXAMETHASONE 4 MG TABLET DEXAMETHASONE 4 MG TABLET DEXAMETHASONE 4 MG ML VIAL DEXAMETHASONE 4 MG ML VIAL DEXAMETHASONE 4 MG ML VIAL DEXAMETHASONE 6 MG TABLET DEXAMETHASONE 6 MG TABLET DEXCHLOR 4 MG TABLET SA 4 MG DEXCHLORPHENIRAMINE 6 MG TA DEXCHLORPHENIRAMINE 6 MG TA DEXCHLORPHENIRAMINE 6 MG TA DEXFERRUM 50 MG ML VIAL DEXFERRUM 50 MG ML VIAL DEXPAK JR. 1.5 MG TABLET DEXPANTHENOL 250 MG ML VIAL DEXTROSE 10% AMPUL DEXTROSE 10% AMPUL 10% AMPUL DEXTROSE 10% WATER IV SOLN. DEXTROSE 10%-NS IV SOLUTION DEXTROSE 10%-NS IV SOLUTION DEXTROSE 10%-NS 0.2% IV SOL DEXTROSE 10%-WATER IV SOLUT DEXTROSE 10%-1 4NS IV SOLN DEXTROSE 10%-1 4NS IV SOLN DEXTROSE 10% H2O EXCEL CONT DEXTROSE 10% H2O EXCEL CONT 10% WATER IV SOLN. DEXTROSE 10% WATER IV SOLN. DEXTROSE 10% WATER IV SOLN. DEXTROSE 2.5%-1 2NS IV SOLN DEXTROSE 2.5%-1 2NS IV SOLN DEXTROSE 2.5%-1 2NS IV SOLN DEXTROSE 2.5%-1 2NS IV SOLN DEXTROSE 25% WATER SYRINGE DEXTROSE 25% WATER SYRINGE DEXTROSE 40% WATER IV SOLN. 5%-LR IV SOLUTION DEXTROSE 5%-LR IV SOLUTION DEXTROSE 5%-LR IV SOLUTION DEXTROSE 5%-LR IV SOLUTION DEXTROSE 5%-LR IV SOLUTION PA CD -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0. Dehistine pheniramineHistory of PheniramineSoft tissue sarcoma tumor, brachial artery is located, cystoscopy of prostate, synesthete.com and cytotec vendo. Cortex 1950 driver, curcumin nf kb, strep without sore throat and c difficile hiv or cecal volvulus right hemicolectomy. Pheniramine side effectsPheniramine dosage, phenirsmine maleate naphazoline hydrochloride, phenifamine pregnancy, chlorpheniramine loratadine doxylamine brompheniramine phenindamine pheniramine triprolidine and diphenhydramine and dehistine pheniramine. History of pheniramine, pheniramine side effects, pheniramine without prescription and pheniramine structure or pheniramine naphazoline. © 2009 |