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Acute exacerbation 98.5% of participants would continue tiotropium. 98.5% would continue salbutamol but 71.5% would change the method of delivery from metereddose inhaler MDI ; to MDI with spacer or nebuliser, and or increase the dosage. 80% would prescribe an oral steroid, most commonly prednisolone or prednisone 50 mg daily for 12 weeks. 98% would prescribe one or more antibiotics, mostly amoxycillin 69.9% ; or doxycyline 14.8% ; , and usually for 12 weeks. Stable disease n 121 ; 98.3% and 70.2% would continue prescribing tiotropium and salbutamol, respectively. 46.3% would add a long-acting beta2 agonist, either as a single product 15.7% ; or in combination with an inhaled corticosteroid 30.6% ; . When prescribed as a single-ingredient product, about half would review the medication after 48 weeks, but when prescribed as a combination product, most would continue it indefinitely. 50.4% added an inhaled corticosteroid, either as a single product 19.8% ; or in combination with a long-acting beta2 agonist 30.6% ; . When prescribed as a single-ingredient product, most would review the medication after 48 weeks, but when prescribed as a combination product, most would continue it indefinitely.

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Table 2. Postpartum Blood Pressure and Pulse Rate Immediately Postpartum and at Transfer From Labor Delivery Recovery Unit to Postpartum Ward for the 3 Patient Groups, because side effect of prednisolone. Irritable bowel syndrome board - i'm new here 20th february 2007.
In the study, researchers looked at whether giving women with recurrent miscarriages the steroid prednisolone could reduce the elevated unk levels in their endometrium uterine lining and protonix.
Needle aspiration is performed only in unusual cases eg, immunocompromised individuals, those not responding to empiric therapy ; because the bacterial etiology of cellulitis in usual cases is highly predictable sachs, 1990; stevenson, 2005.

Rug abuse is a chronic condition often characterized by remissions and relapses. It remains unclear why abstinence can be so difficult to maintain for individuals who were previously dependent. Craving is a term often used by opiate-dependent individuals to explain their relapses to heroin use, but defining this term rigorously in a scientific context has proved difficult 1, 2 ; . Although the term is not explicitly used in ICD-10 or DSM-IV, both systems refer to "a persistent desire" or "strong desire or sense of compulsion" when defining the dependence syndrome. Cue responsivity or cue exposure has been repeatedly used to study craving. Cue responsivity refers to the ability of situations, drug paraphernalia, and mood states or memories associated with previous drug use to effect the desire to use the drug 3 ; . Responses to such cues can be similar or opposite to drug-induced states or can mimic drug withdrawal and may lead to the subjective experience of craving and or the reinitiation or perpetuation of drug-seeking behavior 4 ; . Cue responsivity has received much attention, because the phenomenon may offer avenues for relapse prevention 5 ; . The neural circuits that mediate cue responsivity and craving may be accessible with functional neuroimaging and theo-dur, because stopping prednisolone. Colitis. Moderate colitis requires treatment using a system corticosteroid combined with an aminosalicylate. Ambulatory patients are usually treated with prednisolone and the starting dose is between 30 to 60 mg day. This dose is gradually tapered until the patient is passing normal bowel movements without blood or urgency. If symptoms improve rapidly, the dose of prednisolone can be tapered by 5 to mg week. Patients who are slower to respond require a more gradual tapering schedule, but continuation of high-dose prednisolone must be avoided. If tapering of steroids seems to be difficult, alternative therapy must be taken into consideration. Patients with severe or fulminant colitis require hospitalization and parenteral steroids. Acutely ill patients can be treated with high doses of intravenous corticosteroids for 3 to 5 days, such as methylprednisolone pulse therapy. Mesenteric artery infusion of corticosteroids is effective for some patients, but consultation with a surgeon about colectomy is necessary in such cases. 3. Immunomodulators In the majority of patients, remission is obtained with aminosalicylates and corticosteroids. If it is impossible to taper corticosteroids or frequent relapses occur, immunomodulating therapy should be considered. However, the use of immunomodulators is not approved by the national health insurance scheme, so Japanese physicians are not so experienced with these agents. There are still some specialists who affirm that immunomodulators are ineffective for the treatment of UC while causing serious toxicities. Our experience shows that administration of low-dose azathioprine or 6-MP is beneficial for steroid-dependent UC, but it requires several months for improvement to occur. Reduction of the dose of steroids or even discontinuation may be possible without relapse. Randomized controlled clinical trials have shown strong evidence for the efficacy of these drugs, and they are widely used in Western countries. The major side effects of. Since the total course of prednisolone is for 2-3 months, weekly examination of urine to detect its effect on protein loss is important and ventolin.

In children aged between 1 and 5 years preschool viral wheeze ; transient wheeze triggered by viral colds is common. Most affected children grow out of this by the age of 6 years. Usually symptomatic treatment with bronchodilators is all that is required, however another option is the use of parentalinitiated oral prednisolone at the first sign of symptoms. Although the consensus of opinion in the UK and USA supports this practice, the evidence for this is conflicting. This trial aimed to clarify the situation by evaluating the efficacy of a short course of oral prednisolone for preschool viral wheeze, with stratification for systemic eosinophil priming persistence of wheeze is associated with above-average eosinophil count. However they found no clear benefit of a short course of parent-initiated oral prednisolone for viral wheeze in pre-school children even in those with above-average eosinophil priming Children aged 1-5 years admitted to hospital with viral wheeze, were allocated to either a high-primed or low-primed stratum according to amounts of serum eosinophil cationic protein and eosinophil protein X. They were then randomised in a double blind, cross-over fashion to placebo 108 ; and 109 ; prednisolone 20 mg one daily for 5 days ; for subsequent episodes. Outcome data were available for 120 78% ; of 153 children who had a further episode of viral wheeze, of whom 51 received prednisolone and 69 placebo. Mean daytime difference in means -001 [-022 to 020] ; and night-time 010 [-012 to 032] ; respiratory symptom scores and need for hospital admission did not differ between treatment groups. Within the high-primed n 59 ; and low-primed n 61 ; strata there was no difference in primary outcome between treatment groups. Cochrane Library. 1999; 4: Update Software. 9. Clark P, Tugwell P, Bennet K et al. Injectable gold for rhuematoid arthritis Cochrane Review ; . In: The Cochrane Library. 1999; 4: Update Software. 10. Boers M, Verhoeven A, Markusse H et al. Randomised comparison of combined step-down prednisolone, methotrexate and sulphasalazine with sulphasalazine alone in early rheumatoid arthritis. Lancet. 1997; 350: 309-318. Bresnihan B, Alvaro-Garcia J, Cobby M et al. Treatment of Rheumatoid Arthritis with recombinant human interleukin-1 receptor anatagonist. Arthritis Rheum. 1998; 41: 2196-2204. Forre O, and the Norwegian Arthritis Study Group. Radiologic evidence of disease modification in rheumatoid arthritis patients treated with cyclosporine. Arthritis Rheum. 1994; 37: 1506-1512. Kirwan J, and The arthritis and rheumatism council, low-dose glucocorticoid study group. The effect of glucocorticoids on joint destruction in rheumatoid arthritis . N Engl J Med. 1995; 333: 142-146. Smolen J, Kalden J, Scott D et al. Efficacy and safety of leflunomide compared with placebo and sulphasalazine in active rheumatoid arthritis: a double-blind, randomised, multicentre trial. Lancet. 1999; 353: 259-266. Strand V, Cohen S, Schiff M et al. Treatment of active rheumatoid arthritis with Leflunomide compared with placebo and Methotrexate. Arch Intern Med and cimetidine.

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Prednisone is inactive until converted to prednisolone in the liver. How do soy foods replace unhealthy foods in my diet and differin. The adrenal cortex synthesizes cholesterol and secretes two major corticosteroids: the rnineralocorticoids and the glucocorticoids. Although there is some controversy as to the exact mechanism, glucocorticoids are postulated to suppress the process of IgEmediated bronchoconstriction and to block or inhibit a variety of mediator substances.l, 2, 4, 27 Even though glucocorticoids are not classified as primary bronchodilators, they are known to have the following general effects that contribute to the enhancement of bronchial intraluminal diameteP.27: inhibition of the migration of leukocytes and mast cells reduction of stickiness and margination of polyrnorpholeukocytes potentiation of catecholamine activity reduction of tissue stores of histamine and other mediators suppression of kinin activity, resulting in constriction of the microvasculature stabilization of mast-cell membranes reduction of phosphodiesterase activity It is because of their profound antiinflammatory actions, that glucocorticoids are considered by many to be the drugs of first choice during an acute attack of asthma. The most commonly used glucocorticoids in the treatment of bronchoconstriction are i d e Table 4.ll2z4 in Although it influences all medications, the route of administration of corticosteroids is of particular importance because of the tremendous impact it plays in the incidence of side effectsthe more localized the area of administration, the less likely the chance of systemic reaction. During episodes of severe bronchoconstriction, glucocorticoids are usually administered intravenously eg, methylprednisolone ; . For.

It works by inhibiting the production of allergic and inflammatory mediators in solone omnacortil , prednisolone , delta-cortef , prelone ; take with food or immediately after a meal to prevent stomach upset and eldepryl. A diagnosis of predominantly bulbar type motor neurone disease was made. Topical and oral steroids together with azathioprine 2 mg kg ; and mesalazine suppositories 500 mg bd ; induced a remission of his colitis within three months but the neurological deficit progressively worsened. Subsequently, riluzole 100 mg daily ; a drug which presynaptically inhibits the release of glutamic acid in the central nervous system was commenced and the patient reported that his tongue was more mobile. He defaulted treatment for 18 months before seeking medical attention again recently, whence he displayed extreme emotional lability, was only able to utter incomprehensible words and had significantly more muscle wasting of both upper and lower limbs. He had been self-medicating with prednisolone 10 mg po daily to keep his colitis at bay. DISCUSSION Various neurological complications have been reported in association with ulcerative colitis; albeit in a sporadic fashion. These include thromboembolic disease causing cerebrovascular disorders, neuropathy usually as an acute or chronic inflammatory demyelinating polyneuropathy ; which occurs mostly in ulcerative colitis and myopathies and myelopathies which are more characteristic of Crohn's disease 1-7 ; . There is a concurrence of multiple sclerosis and inflammatory bowel disease both within families and within individuals 8-10 ; . To the best of our knowledge, this is the first report of motor neurone disease complicating ulcerative colitis. This link may be coincidental, related to the basic disease or to its complications or treatment. Complications and extraintestinal manifestations may precede or follow the diagnosis of IBD and may occur with exacerbations of bowel symptoms or independently 11 ; . The pathogenesis of neurological complications in inflammatory bowel disease is largely unknown though coagulation abnormalities, vasculitis, factor V Leiden mutation, circulating immune complexes, autoimmunity, and an infective aetiology e.g. Campylobacter jejuni infection causing an acute inflammatory demyelinating polyradiculoneuropathy ; have been implicated 1, 12-16 ; . The prevalence of autoimmune disorders is three times greater than expected in patients with ulcerative colitis 17 ; . In this patient, the absence of autoantibodies and the failure of immunosuppressive therapy to halt the progression of neurological deficit argue against an autoimmune hypothesis. An underlying vasculitis is unlikely, in view of a normal ESR and continued neurological deterioration despite steroid and azathioprine therapy. These antibodies are stable for at least o o one 1 ; year at -20 C to -70 C. Store product in appropriate aliquots to avoid multiple freeze-thaw cycles. For in vitro investigational use only. Not for use in therapeutic or diagnostic procedures and feldene. A-SMART PremierTM Cart Systems . Contact: Armstrong Medical Ind. Inc. A-SMART Standard Cart Systems . Contact: Armstrong Medical Ind. Inc. A.I.R.S Contact: Omron Healthcare Inc. A.R.M. Allergy Relief Medicine . Contact: B. F. Ascher & Co., Inc. AAD . Contact: Profile Respiratory Systems Ltd. AARC Times . Contact: Daedalus Enterprises Inc. AARC Times . Contact: American Association for Respiratory Care ABLTM . Contact: Radiometer America Inc.

Henry D. Royal, MD, immediate past-chair of the Guidelines and Communications Committee, Commission on Health Care Policy and Practice, for overall coordination and oversight of the Society of Nuclear Medicine Guideline Development Project; Marie Davis, Division of Health Care Policy, Society of Nuclear Medicine, for project coordination, data collection and editing; Wendy J.M. Smith, MPH, Director, Director of Health Care Policy, Society of Nuclear Medicine, for project supervision; and members of the Guideline Development Subcommittee, who contributed their time and expertise to the development of this information and frusemide. 327. Kallen B, Rydhstroem H, Aberg A. Asthma during pregnancy-a population based study. Eur J Epidemiol 2000; 16: 167-71. Cydulka RK, Emerman CL, Schreiber D, et al. Acute asthma among pregnant women presenting to the emergency department. J Respir Crit Care Med 1999; 160: 887-92. Department of Health. Why mothers die. Confidential enquiries into maternal deaths in the United Kingdom 1994-96. London: The Stationery Office; 1998. [cited 17 Jul 2002]. Available from url: : archive.official-documents document doh wmd wmdhm 330. Lewis G, editor. Why mothers die 1997-1999. The fifth report of the confidential enquiries into maternal deaths in the United Kingdom 1997-99. London: RCOG Press; 2001. [cited 17 Jul 2002]. Available from url: : cemd cemdrpt 331. Schatz M, Zeiger RS, Harden K, et al. The safety of asthma and allergy medications during pregnancy. J Allergy Clin Immunol 1997; 100: 3016. Rayburn WF, Atkinson BD, Gilbert K, et al. Short-term effects of inhaled albuterol on maternal and fetal circulations. J Obstet Gynecol 1994; 171: 770-3. Schatz M, Zeiger RS, Harden KM, et al. The safety of inhaled betaagonist bronchodilators during pregnancy. J Allergy Clin Immunol 1988; 82: 686-95. Mann RD, Kubota K, Pearce G, et al. Salmeterol: a study by prescriptionevent monitoring in a UK cohort of 15, 407 patients. J Clin Epidemiol 1996; 49: 247-50. Greenberger PA, Patterson R. Beclomethasone diproprionate for severe asthma during pregnancy. Ann Intern Med 1983; 98: 478-80. Dombrowski M, Thom E, McNellis D. Maternal-Fetal Medicine Units MFMU ; studies of inhaled corticosteroids during pregnancy. J Allergy Clin Immunol 1999; 103: S356-9. 337. Dombrowski MP, Brown CL, Berry SM. Preliminary experience with triamcinolone acetonide in pregnancy. J Matern Fetal Med 1996; 5: 3103. Kallen B, Rydhstroem H, Aberg A. Congenital malformations after the use of inhaled budesonide in early pregnancy. Obstet Gynecol 1999; 92: 292-5. Stenius-Aarniala B, Riikonen S, Teramo K. Slow-release theophylline in pregnant asthmatics. Chest 1995; 107: 642-7. Schatz M. Asthma during pregnancy: interrelationships and management. Ann Allergy 1992; 68: 123-33. Czeizel AE, Rockenbauer M. Population-based case-control study of teratogenic potential of corticosteroids. Teratology 1997; 56: 335-40. Park-Wyllie L, Mazzotta P, Pastuszak A, et al. Birth defects after maternal exposure to corticosteroids: prospective cohort study and meta-analysis of epidemiological studies. Teratology 2000; 62: 385-92. Rodriguez-Pinilla E, Martinez-Frias ML. Corticosteroids during pregnancy and oral clefts: a case-control study. Teratology 1998; 58: 25. The use of newer asthma and allergy medications during pregnancy. The American College of Obstetricians and Gynecologists ACOG ; and The Americal College of Allergy, Asthma and Immunology ACAAI ; . Ann Allergy Asthma Immunol 2000; 84: 475-80. Mabie WC, Barton JR, Wasserstrum N, et al. Clinical observations on asthma in pregnancy. Obstet Gynecol Surv 1992; 47: 464-6. Lao TT, Huengsburg M. Labour and delivery in mothers with asthma. Eur J Obstet Gynecol Reprod Biol 1990; 35: 183-90. Arad I, Landau H. Adrenocortical reserve of neonates born of long-term, steroid-treated mothers. Eur J Pediatr 1984; 142: 279-80. Gruskay FL. Comparison of breast, cow, and soy feedings in the prevention of onset of allergic disease: a 15-year prospective study. Clin Pediatr Phila ; 1982; 21: 486-91. Saarinen UM, Kajosaari M. Breastfeeding as prophylaxis against atopic disease: prospective follow-up study until 17 years old. Lancet 1995; 346: 1065-9. Turner ES, Greenberger PA, Patterson R. Management of the pregnant asthmatic patient. Ann Intern Med 1980; 93: 905-18. Ost L, Wettrell G, Bjorkhem I, et al. Predbisolone excretion in human milk. J Paediatr 1985; 106: 1008-11. McKenzie SA, Selley JA, Agnew JE. Secretion of prednisolobe into breast milk. Arch Dis Child 1975; 50: 894-6. Greenberger PA, Odeh YK, Frederiksen MC, et al. Pharmocokinetics of prdnisolone transfer to breast milk. Clin Pharmacol Ther 1993; 53: 3248. Meredith S, Nordman H. Occupational asthma: measures of frequency from four countries. Thorax 1996; 51: 435-40. Ross DJ. Ten years of the SWORD project. Surveillance of Work-related and Occupational Respiratory Disease. Clin Exp Allergy 1999; 29: 7503. Hendrick DJ, Burge PS. Asthma. In: Hendrick DJ, Beckett W, Burge PS, Churg A, editors. Occupational disorders of the lung. Recognition, management and prevention. London: WB Saunders; 2002. p. 33-76.

Of the Alabama Department and services to mentally of the state. institution operating under the of Mental Health. It provides ill persons from the upper and keflex and prednisolone, for instance, predniwolone allergy.

Hey this might sound stupid but i live in the uk and we only get methylprednisolone as an what is it. What medications are you currently taking? Do you draw a blank when asked this question at your doctor's visit? Creating a personal medication record is a convenient way to keep track of any over-the-counter drugs, prescription medications or dietary supplements you may be taking. Carry this wallet-sized list to medical appointments to help you provide an accurate report of your current medications. Request a free medication record by returning the postage-paid postcard on the back panel of this newsletter and nifedipine. Gilman MJ, Meyer L, Carter J, Slovis C. Comparison of aerosolized glycopyrrolate and metaproterenol in acute asthma. Chest 1990; 98: 10951098. Walker FB, Kaiser DL, Kowal MB, Suratt PM. Prolonged effect of inhaled glycopyrrolate in asthma. Chest 1987; 91: 4951. Johnson BE, Suratt PM, Gal TJ, Wilhoit SC. Effect of inhaled glycopyrrolate and atropine in asthma. Chest 1984; 85: 325328. Schroeckestein DC, Bush RK, Chervinsky P, Busse WW. Twelve hour bronchodilation in asthma with a single aerosol dose of the anticholinergic compound glycopyrrolate. J Allergy Clin Immunol 1988; 82: 115119. Lung function testing: selection of reference values and interpretative strategies. Rev Respir Dis 1991; 144: 12021218. Dixon WJ Ed. ; . BMDP statistical software manual. Berkeley, CA, University of California Press, 1990; Vol. 2. Komanapolli S, Fulambarker A, Tyler D, Vega D, Patel J, Tzelepis G. Effects of glycopyrrolate and metaproterenol alone and in combination in COPD. Rev Respir Dis 1993; 147 4 ; : A316 Abstract ; . Dougals NJ, Davidson I, Sudlow MF, Fleney D. Bronchodilatation and the site of airway resistance in severe chronic bronchitis. Thorax 1979; 34: 5156. Lightbody LM, Ingram CG, Legge JS, Johnston RN. Ipratropium bromide, salbutamol and prednisolone in bronchial asthma and chronic bronchitis. Br J Dis Chest 1978; 72: 181186. Koyama H, Nishimura K, Ikeda A, Izumi T. A comparison of the bronchodilating effects of oxitropium bromide and fenoterol in patients with chronic obstructive pulmonary disease. Chest 1993; 104: 17431747. Less AW, Allan GW, Smith J. Nebulized ipratropium bromide and salbutamol in chronic bronchitis. Br J Clin Pract 1980; 3: 340342. Leitch AG, Hopkin JM, Ellis DA, Merchant S, McHardy.

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D. Long-Term Care Pharmacies Under the MMA, Congress directed CMS to establish rules governing "convenient access" for pharmacies in a PDP's network, and authorized but did not direct ; CMS to consider developing "standards with respect to access" for long-term care facility residents.35 CMS understood that the long-term care pharmacy industry was somewhat unique in the sense that most long-term care residents received their medication through one long-term care pharmacy under contract with the facility. This would present a problem if an individual enrolled with a PDP and was later admitted to a long-term care facility. If that PDP did not have a contract with the long-term care pharmacy, the individual would be forced to go out-of-network i.e., to the long-term care pharmacy ; for drugs, a thoroughly unappealing prospect given that the individual would be financially responsible for the cost of the drugs. CMS initially considered requiring or strongly encouraging ; PDPs to offer their standard contract to all or some longterm care pharmacies in their service areas. In addition, the agency considered allowing long-term care residents to, in fact, go out of network for drugs in longterm care facilities. CMS also solicited comments on how to ensure access for long-term care facility residents. CMS received several comments in response.36 The final rule adopts a free-market approach to the question of long-term care pharmacies; CMS says that it is "essential to inject competition into the long-term care pharmacy market."37 At the same time, the regulation contains protection for beneficiaries by allowing a special enrollment period for a beneficiary who is about to enter a long-term care facility.38 With regard to competition in the marketplace, CMS has established an "any willing pharmacy" requirement for PDPs to develop relationships with longterm pharmacies. At the same time, CMS has coupled that provision with an express invitation for any pharmacy in a PDP service area to bid to become a long-term pharmacy by meeting certain patient protection criteria.39 As a result, it is possible that long-term care facilities will be serviced by several long-term care.

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Ollowing soon after the recent approval of teriparatide rhPTH[134] ; by Health Canada, the Fall issue of Osteoporosis Update discusses this new, much-anticipated osteoporosis therapy. The first in a novel class of anabolic treatments, teriparatide acts to improve skeletal microarchitecture and increase bone mineral density BMD ; by stimulating new bone formation. Individuals who we hope will benefit from this option include postmenopausal women as well as men with severe osteoporosis, known to have already suffered a fragility fracture. Additional candidates for teriparatide therapy include patients not responding to antiresorptive agents currently in use, and those with established glucocorticoid-induced osteoporosis who require long-term steroid treatment. The addition of teriparatide to our menu of available osteoporosis treatment, because prednisolone uses. Naunyn schmiedebergs arch pharmacol 347 : 79-8 1993 and protonix.

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Bellet PS, Kalinyak KA, Shukla R, Gelfand MJ, Rucknagel DL. Incentive spirometry to prevent acute pulmonary complications in sickle cell diseases. N Engl J Med 1995; 333 11 ; : 699-703. Griffen TC, McIntire D, Buchanan GR. High-dose intravenous methylprednisolone therapy for pain in children and adolescents with sickle cell disease. N Engl J Med 1994; 330 11 ; : 733-737. Jacobson SJ, Kopecky EA, Joshi P, Babul N. Randomised trial of oral morphine for painful episodes of sickle-cell disease in children. Lancet 1977; 350: 1358-1361.
Haisch, A., A. Groger, et al. 2000 ; . "[Protection of autogenous cartilage transplants from resorption using membrane encapsulation]." Hno 48 2 ; : 119-24. Haisch, A., S. Klaring, et al. 2002 ; . "A tissue-engineering model for the manufacture of auricular-shaped cartilage implants." Eur Arch Otorhinolaryngol 259 6 ; : 316-21. Haisch, A., O. Schultz, et al. 1996 ; . "[Tissue engineering of human cartilage tissue for reconstructive surgery using biocompatible resorbable fibrin gel and polymer carriers]." Hno 44 11 ; : 624-9. Haisch, A., F. Wanjura, et al. 2004 ; . "Immunomodulation of tissue-engineered transplants: in vivo bone generation from methylprednisolone-stimulated chondrocytes." Eur Arch Otorhinolaryngol 261 4 ; : 216-24. Han, B., I. R. Schwab, et al. 2002 ; . "A fibrin-based bioengineered ocular surface with human corneal epithelial stem cells." Cornea 21 5 ; : 505-10. Hartig, G. K., R. M. Esclamado, et al. 1994 ; . "Comparison of the chondrogenic potential of free and vascularized perichondrium in the airway." Ann Otol Rhinol Laryngol 103 1 ; : 9-15. Harvey, S. 1916 ; . "The use of fibrin paper and forms in surgery." Boston Med Surg J 174: 658-659. Hegewald, A. A., J. Ringe, et al. 2004 ; . "Hyaluronic acid and autologous synovial fluid induce chondrogenic differentiation of equine mesenchymal stem cells: a preliminary study." Tissue Cell 36 6 ; : 431-8. Heystek, J., G. M. Maier-van der Zande, et al. 1975 ; . "Contributions to the optimal use of human blood. VI. Modification of the method to prepare prothrombin complex on a large scale." Vox Sang 29 3 ; : 177-83. Hino, M., O. Ishiko, et al. 2000 ; . "Transmission of symptomatic parvovirus B19 infection by fibrin sealant used during surgery." Br J Haematol 108 1 ; : 194-5. Hino, M., R. Yamamura, et al. 1999 ; . "[Human parvovirus B19-induced aplastic crisis in a patient treated with fibrin sealant]." Rinsho Ketsueki 40 2 ; : 145-9. Homminga, G. N., P. Buma, et al. 1993 ; . "Chondrocyte behavior in fibrin glue in vitro." Acta Orthop Scand 64 4 ; : 441-5. Hunter, C. J., S. M. Imler, et al. 2002 ; . "Mechanical compression alters gene expression and extracellular matrix synthesis by chondrocytes cultured in collagen I gels." Biomaterials 23 4 ; : 1249-59. Hunter, C. J. and M. E. Levenston 2002 ; . "The influence of repair tissue maturation on the response to oscillatory compression in a cartilage defect repair model." Biorheology 39 1-2 ; : 79-88. Hunter, C. J. and M. E. Levenston 2004 ; . "Maturation and integration of tissue-engineered cartilages within an in vitro defect repair model." Tissue Eng 10 5-6 ; : 736-46. Hutmacher, D. W., J. C. Goh, et al. 2001 ; . "An introduction to biodegradable materials for tissue engineering applications." Ann Acad Med Singapore 30 2 ; : 183-91. Israels, S. J. and E. D. Israels 1994 ; . "Development of antibodies to bovine and human factor V in two children after exposure to topical bovine thrombin." J Pediatr Hematol Oncol 16 3 ; : 249-54. Israels, S. J. and M. T. Leaker 1997 ; . "Acquired inhibitors to factors V and X after exposure to topical thrombin: interference with monitoring of low molecular weight heparin and warfarin." J Pediatr 131 3 ; : 480-3. Kamil, S. H., R. D. Eavey, et al. 2004 ; . "Tissue-engineered cartilage as a graft source for laryngotracheal reconstruction: a pig model." Arch Otolaryngol Head Neck Surg 130 9 ; : 1048-51. Kamil, S. H., K. Kojima, et al. 2003 ; . "In vitro tissue engineering to generate a human-sized auricle and nasal tip." Laryngoscope 113 1 ; : 90-4.

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