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TABLES. iii FIGURES. vii ACKNOWLEDGEMENTS . viii 1. BACKGROUND . 1 2. METHODS . 2 2.1 Population . 2 2.2 Data. 3 2.2.1 Case and resource use data . 3 2.2.2 Unit cost data. 5 2.3 Treatment Analysis . 7 2.4 Cost Analysis . 8 3. TREATMENT PATTERNS. 10 3.1 Manitoba Health databases . 10 3.1.1 Medical claims . 10 3.1.2 Hospitalizations . 13 3.1.3 Prescription Drugs . 18 3.2 OpTx and chart review . 20 3.2.1 Physician visits . 21 3.2.2 Procedures . 23 3.2.3 Laboratory tests . 23 3.2.4 Radiation therapy . 26 3.2.5 Drugs. 29 3.2.6 Outpatients. 31 3.2.7 Emergency visits . 32 3.3 Treatment summary . 34 4. COST PATTERNS . 35 4.1 Manitoba Health Databases and OpTx . 35 4.1.1 Numbers of services and costs by treatment type . 35 4.1.2 Average cost per patient-month constant prices and discounted . 37 4.1.3 Average cost per patient-month by month of follow-up. 37 4.1.4 Lifetime treatment costs . 46 4.2 Chart Review . 47 4.2.1 Costs of additional health services . 47 4.2.2 Average cost per patient-month. 52 4.2.3 Lifetime treatment costs . 59 5. DISCUSSION and CONCLUSION. 61 6. REFERENCES. 66 APPENDICES . 68, for example, benazepril hcl 40 mg.

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1. NHLBI, National Institutes of Health. Morbidity & Mortality: 1998 Chartbook on Cardiovascular, Lung and Blood Diseases. October 1998. 2. National Institutes of Health, National Institute of Neurological Disorders and Stroke. Stroke: Hope Through Research. ninds.nih.gov, May 1999. 3. Karem JH. Diabetes mellitus In: Tierney LH, McPhee SJ, Papdakis MA eds. Current Medical Diagnosis and Treatment. Stamford, Conn: Appleton and Lange; 1996. 4. American Medical Association. The Sixth Report of the Joint National Committee on Prevention, Detection, Evaluation and Treatment of High Blood Pressure. Arch Inter Med. 1997; 157: 2413-2446 Wolf PA, D'Agostino RB, Belanger AJ, Kannel WB: Probability of stroke: a risk profile from The Framingham Study. Stroke. 1991; 22: 312-318. Kannel Wb, Abbott RD, Savage DD, McNamara PM. Epidemiologic features of chromic atrial fibrillation, : The Framingham Study. N. Eng. J. Med. 1982; 306: 1018-1022. Wolf PA, Doubler TR, Thomas HE, et al. Epidemiologic assessment of chronic atrial fibrillation and risk of stroke: The Framingham Study. Neurol. 1987; 28: 973-977. Wolf PA, Abbott RD, Kannel WB. Atrial Fibrillation: A major contributor to stroke in the elderly: The Framingham Study. Arch Inter Med. 1987; 147: 1561-1564. Sacco RL. Wolf PA, Kannel WB, et al. Survival and recurrence of stroke. The Framingham Study. Stroke. 1982; 13: 290-295. Adapted from Shinton R, Beevers G. Meta-analysis of relation between cigarette smoking and stroke. BMJ. 1989; 298: 789-794. Thornton J, Symes C, Heaton K. Moderate alcohol intake reduces bile cholesterol saturation and raises HDL cholesterol. Lancet. 1983; 2: 819-822. Pellegrini N, Paret Fl, Stabile F Brusamolino A, Simonetti P Effects of moderate , . consumption of red wine on platelet aggregation and hemostatic variables in health volunteers. Eur J Clin Nutr. 1996; 50: 209-213. Smith D. Stroke prevention: the importance of risk factors. Stroke: Clinical Updates. National Stroke Association. 1991; 1: 17-20. Munschauer FE, Hens MM, Priore RL, et al. Screening for atrial fibrillation in the community: A multi-center validation trial. J Stroke and Cerebrovascular Dis. 1999; 8 2 ; : 99-103 15. Petitti DB, Winger J. Use of oral contraceptives and cigarette smoking and risk of subarachnoid hemorrhage. Lancet. 1978; 2 8083 ; : 234-5. 16. Adapted from: MacMahon S. The effects of anti-hypertensive drug treatment on the incidence of stroke and coronary heart disease. Clin Exp Hypertension . 1989; All: 807-823. 17. Adapted from: SHEP Cooperative Research Group. Prevention of stroke by antihypertensive drug treatment in older persons with isolated systolic hypertension: final results of the Systolic Hypertension in the Elderly Program SHEP ; . JAMA. 1991; 265; 3255-3264 and betamethasone, for instance, benazepril feline. Some patients must also give up various medications that interfere with their improving or recovering. There are many different over-the-counter otc ; pain relievers and fever medications and bethanechol.

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M. Goumenou, K. Machera. Laboratory of Pesticides Toxicology, Benaki Phytopathological Institute, Kifissia, Athens, Greece Captan CA ; and carbendazim CB ; are two widely used fungicides applied as individual formulations, as a commercially available premixture or as spray tank mixtures. CB is a well-known aneugen while CA is not considered as an in vivo genotoxic substance. For the study of the in vivo genotoxic potency of the combination of CA and CB formulations CAF, CBF ; as well as their possible interaction, 2 separate experiments were performed using the bone marrow micronucleus test in rats. In both experiments, 3 dose levels of the mixture were studied 977, 1563 and 2500 mg of each active ingredient kg b.w. ; . Sampling time for all test groups was 24h while for the highest dose group samples were also taken at 48h. The individual formulations were tested at 2500 mg a.i. kg b.w, at 24 and 48h sampling time. For the study of possible interaction, the dose-response curve of the CBF at the above mentioned dose levels 24h post treatment of male rats ; and the respective curve for the same CBF dose levels with constant addition of the CAF at 2500 mg a.i. kg b.w were established. Regarding the MN incidence in comparison to the control group, a significant increase was observed in all CBF groups at 24 and 48h. A slight, statistically non-significant increase at 24h in CAF alone and a marginal increase in some combination groups were observed. CBF and the combination were cytotoxic at 48h while CAF was cytotoxic at both 24 and 48h. The dose-response curve for micronuclei formation following CBF administration was well fitted to the cubic model. Comparing this curve with the curve established after constant addition of 2500 mg a.i. kg b.w. of CAF to the CBF doses, a considerable reduction of the MN incidence induced from CBF was observed. Additionally, the MN incidence from mixture groups with different composition from those that formed the above-mentioned curve was lower than every CBF dose group. Consequently, the 2 fungicide formulations seem to interact resulting in a reduction of the CBF genotoxicity. 552 and zebeta.

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