In several clinical studies ondansetron has been shown to be effective for the treatment of emesis, in particular nausea and vomiting associated with cancer chemotherapy and radiotherapy and post-operatively occurring nausea.
Ondansetron HCl Tab 8mg Ondanseton HCl Oral Soln 4mg 5ml S F Indansetron HCl Suppos 16mg Zofran Tab 4mg Zofran Tab Melt 4mg Prochlpzine Mal Suppos 5mg Prochlpzine Mal Suppos 25mg Prochlpzine Mal Tab 5mg Prochlpzine Mal Tab 25mg Prochlpzine Mal Tab Buccal 3mg Stemetil Tab 5mg Stemetil Suppos 5mg Stemetil Suppos 25mg Buccastem Tab 3mg Proziere Tab 5mg Buccastem M Tab 3mg Prochlpzine Mesil Oral Soln 5mg 5ml Prochlpzine Mesil Inj 12.5mg ml 1ml Amp Prochlpzine Mesil Gran Sach Eff 5mg S F Stemetil Syr 5mg 5ml Stemetil Inj 1.25% 12.5mg 1ml Amp Stemetil Eff Gran Sach 5mg Lem S F Promethazine Teoclate Tab 25mg Avomine Tab 25mg Aspav Disper Tab Aspirin Tab E C 300mg Aspirin Disper Tab 300mg Aspirin Tab 300mg Caprin Tab E C 300mg Nu-Seals 300 Tab E C 300mg Co-Codamol Tab 8mg 500mg Co-Codamol Cap 8mg 500mg Co-Codamol Eff Tab 8mg 500mg Co-Codamol Cap 30mg 500mg Co-Codamol Eff Tab 30mg 500mg Co-Codamol Tab 30mg 500mg.
TABLE 1. CONCLUSIONS 1. Risk factors for coronary and non-coronary atherosclerotic disease are generally similar and independent of the end organ subserved by a given arterial vascular bed. Risk factors that are modifiable include elevated lipid levels, cigarette smoking, systolic and diastolic hypertension, diabetes mellitus, sedentary life style, psychosocial factors and obesity. 2 Identification and modification of established risk factors are important in reducing the risk of developing PAD and reducing the risk of adverse cardiovascular events. 3 Promising emerging risk factors or markers are being evaluated for their utility in predicting prognosis and stratifying of risk. There is inadequate information to support routine measurements of these markers.
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CHERTKOW Howard Local: New treatments for Alzheimer's disease: looking for miracles, settling for less? Grand Medical Rounds, Department of Medicine, Jewish General Hospital, Montreal: May, 1998, because ondansetron hcl 8.
Oral administration of ondansetron up to 15 mg kg day did not affect fertility or general reproductive performance of male and female rats!
ANTI-EMETIC DRUGS Metoclopramide 10mg in 2ml injection Prochlorperazine 12.5mg in 1ml injection by deep im injection only; do not administer intravenously or subcutaneously ; Cyclizine 50mg as a bolus or added to PCA morphine syringe. Oondansetron 4mg in 2ml IV injection to be used for resistant post operative nausea and vomiting PONV ; , or in children with PONV - dose 0.1mg kg and zofran.
| Ondansetron more drug_usesMinerals calcium is found in milk, cheese, nuts, pulses, green leafy vegetables, tinned sardines and salmon and sesame seeds and helps to maintain healthy bones and teeth.
Month period. 6 In contrast, for patents not listed in the Orange Book, a patent holder must sue a generic company for patent infringement in the district courts based on ordinary federal litigation procedures, without the benefit of a 30-month stay. Under non-Hatch-Waxman litigation, the branded firm must obtain a preliminary injunction to prevent the sale of the generic product before the conclusion of the suit. The operation of this 30- month stay under the Hatch-Waxman Amendments is the FTC's primary concern with regard to "Orange Book" listings. 7 The FTC believes the process is subject to abuses that extend the patent monopoly substantially beyond the terms of the original patent. The FTC's generic drug study examined these issues in detail and provided much of the empirical data supporting the FTC's enforcement policy against improper Orange Book listings and oxcarbazepine, for instance, ondansetron and pregnancy.
See useful links page for the address of the various medical boards in australia to confirm your state requirements please contact the relevant state medical board to confirm the exact current paperwork required.
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Disease in newly diagnosed type 2 non-insulin-dependent ; diabetes. Diabetologia 1985; 28: 653-9. Garcia-Webb P. Bonser AM, Whiting D, Masarel JRL. Insulin resistance-a risk factor for coronary heart disease? Scand J Clin Lab Invest 1983; 43: 677-85. World Health Organization. WHO Study Group on diabetes mellitus. Geneva: WHO 1985. Technical report series 727. ; 12. Grundy SM, Nix D, Whelan MF, Franklin L Comparison of three cholesterol-lowering diets in normolipidemic men. JAMA and
trileptal.
The ideal drug to treat inflammatory disease would be able to both blunt the inflammatory response and activate the resolution of inflammation.
Adverse effects: ondansetron has been administered to over 2500 patients worldwide in controlled clinical trials and has been well tolerated and oxytetracycline.
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Postoperative nausea and vomiting PONV ; are undesirable outcomes from surgery that can lead to electrolyte abnormalities, dehydration, pulmonary aspiration, wound dehiscence, and other complications. These complications can prolong the recovery room stay and result in unanticipated hospitalization of ambulatory surgery patients. Up to 50% of patients undergoing ambulatory surgery experience PONV. Risk factors for PONV include female gender, nonsmoking status, history of PONV or motion sickness, a long duration of surgery, the type of surgery, and the intraoperative or postoperative use of opioids. Various peripheral and central neurotransmitters in the gastrointestinal GI ; tract and brain stem are thought to mediate PONV. The effects of most antiemetic drugs that have been used to prevent or treat PONV are mediated by blocking dopamine type 2, serotonin type 3, histamine type 1, or muscarinic cholinergic type 1 receptors. The usefulness of these agents e.g., scopolamine, dimenhydrinate, prochlorperazine, promethazine, ondansetron ; is sometimes limited by adverse effects and a lack of efficacy. Research has focused on substance P, a neuropeptide that interacts with neuokinin-1 NK-1 ; receptors in the GI tract and brainstem and is thought to play an important role in PONV. Aprepitant, the first agent in a new class of antiemetic drugs known as NK-1 receptor antagonists, was introduced initially for the prevention of chemotherapy-induced nausea and vomiting and it recently was approved for the prevention of PONV. The use of combination antiemetic therapy that inhibits more than one neurotransmitter involved in PONV may prove more effective than monotherapy in patients at high risk for PONV.
Albers Medical Distributors, Inc., paid defendant Paul Louis Kriger approximately $106, 176.96 in payment for his role in facilitating the purchase and sale of the stolen Glaxo drugs. Overt Acts - Stolen Roche Drugs 1. In or about June 2002, Julio Cesar Cruz acquired control over the stolen Roche drugs and
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Pharmacy & Therapeutic Committee Emend aprepitant ; 7 2003 Recommendations: P&T MEC approved MRMC RCH SMH Oral Aprepitant EmendTM ; is recommended for addition to the formulary. Oral Aprepitant, in combination with a 5HT3 antagonist and dexamethasone, is FDA approved for prevention of acute and delayed nausea and vomiting associated with initial and repeat courses of highly emetogenic cancer chemotherapy including high dose cisplatin ; . The FDA recommended dosage of aprepitant, 1 hour before chemotherapy, is 125mg orally on day 1 and 80mg orally on days 2 3, along with a 5HT3 receptor antagonist on day 1, and dexamethasone on days 1-4. Granisetron 2 mg PO and dexamethasone PO are recommended to be given in combination Aprepitant as efficacy is unchanged and cost is reduced by using the oral route. The American Society of Clinical Oncology recommends the oral route. Orders will be entered for the oral route unless the patient is unable to use oral route. Findings: Aprepitant, in combination with other antiemetics agents, is FDA approved for prevention of acute and delayed nausea and vomiting associated with initial and repeat courses of highly emetogenic cancer chemotherapy including high dose cisplatin ; . Chronic continuous use of aprepitant is not recommended. Aprepitant is a highly selective antagonist of human substance P neurokinin 1 NK1 ; receptors. Aprepitant crosses the blood brain barrier and occupies the NK1 receptors. The NK1 receptors mediate the biological activities of Substance P. Substance P and the receptors are located in the brainstem dorsal vagal complex and in the GI tract. Chemotherapeutic agents activate the Chemoreceptor trigger zone CTZ ; by releasing various neurotransmitters including Substance P ; both peripherally and centrally to induce chemo nausea and vomiting. Aprepitant has little or no affinity for serotonin 5-HT3 ; , dopamine, and corticosteroid receptors. Aprepitant is eliminated primarily by metabolism and is not renally excreted. Therefore, no dosage adjustment is necessary in the elderly, patients with mild to moderate hepatic insufficiency Child-Pugh score of 5-9 ; , patients with renal insufficiency CrCl 30mL min ; , and patients with ESRD undergoing hemodialysis. Studies have not be conducted in patients with Child-Pugh scores 9. Aprepitant is a substrate, a moderate inhibitor, and an inducer of CYP3A4. It should not be used concurrently with pimozide, terfenadine, astemizole, or cisapride. Aprepitant is an inducer of CYP2C9, increases the metabolism of warfarin and tolbutamide. Interactions with granisetron or ondansetron have not been demonstrated. Chemotherapy agents that are known to be metabolized by CYP3A4 include docetaxel, paclitaxel, etoposide, irinotecan, ifosfamide, imatinib, vinorelbine, vinblastine, and vincristine. In clinical studies aprepitant was commonly administered with etoposide, vinorelbine, or paclitaxel. The doses of these agents where not altered to account for potential drug interactions. Small number of patients in clinical studies received docetaxel, vinblastine, vincristine or ifosfamide. Dosage and administration: 125 mg PO 1 hours before chemotherapy on day 1, 80 mg PO 1 hour before chemotherapy on days 2 and 3. Aprepitant Package Insert Study One Two ; Aprepitant Standard Therapy N 260 N 261 Complete Response * Overall 73% 63% 52% % Acute Phase 89% 83% 78% Delayed Phase 75% 68% 56% Complete Protection + Overall 63% 56% 49% Acute Phase 85% 80% 75% Delayed Phase 66% 61% 52% * Complete Response: No emetic episodes & no use of rescue therapy + Complete Protection: No emetic episodes, no use of rescue therapy, and a maximum nausea visual analog scale 25 mm on 0-100 scale.
Bermudez J, Boyle EA, Minter WD, Sanger GJ. The antiemetic potential of the 5-hydroxytryptamme, receptor antagonist BRL 43694. Br J Cancer 1988; 58: 644-50. Andrews PLR, Bhandari P, Davey PT, et al. Are all 5-HT3 receptor antagonists the same? Eur J Cancer 1992; 28: S2-6. Fujii Y, Tanaka H, Toyooka H. Reduction of postoperative nausea and vomiting with granisetron. Can J Anaesth 1994; 41: 291-4. Fujii Y, Tanaka H, Toyooka H. Optimal anti-emetic dose of granisetron for preventing postoperative nausea and vomiting. Can J Anaesth 1994; 41: 794-7. Watcha MF, White PF. Postoperative nausea and vomiting: its etiology, treatment, and prevention. Anesthesiology 1992; 77: 162-84. Maisano R, Adam0 V, Settineri N, et al. Efficacy of two oral dose regimens of granisetron. Anticancer Res 1995; 15: 2287-90. Fujii Y, Tanaka H, Toyooka H. Preoperative oral granisetron prevents postoperative nausea and vomiting. Acta Anaesthesiol Stand 1998; 42: 653-7. Brogden RN, Carmine AA, Heel RC, et al. Domperidone: a review of its pharmacological activity, pharmacokinetics and therapeutic efficacy in the symptomatic treatment of chronic dyspepsia and as an antiemetic. Drugs 1982; 24: 360-400. Carmichael J, Cantwell BMJ, Edwards CM, et al. A pharmacokinetic study of granisetron BRL 43694A ; , a selective 5-HT, receptor antagonist: correlation of anti-emetic response. Cancer Chemother Pharmacol 1989; 24: 45-9. Kudoh S, Okada H, Miyazaki R, et al. A phase I study of granisetron hydrochloride: pharmacokinetics of granisetron following single oral administration in healthy Japanese subjects. Jpn Pharmacol Ther 1993; 21: 169-76. Aspinall RL, Goodman NW. Denial of effective treatment and poor quality of clinical information in placebo controlled trials of ondansetron for postoperative nausea and vomiting: a review of published trials. BMJ 1995; 311: 844-6 and
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I wrote to the fda and i've not received a reply, which i was assured would be forthcoming, nothing works to get rid of this toxin and our medical profession does not accept any responsibilty in prescribing this harmful drug, for instance, ondansetron nausea.
A paper in Pharmacotherapy June 2002 ; reported a case of a 58-year old male who presented with a 3-day priapism. He thought he had taken aspirin for a headache and
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OB GYN TOPICAL ANTIINFECTIVES. 52 OBSTETRICAL & GYNECOLOGICAL MEDICATIONS . 50 octreotide . 16, 17 ocusulf. 55 ofloxacin. 11, 55 ogestrel. 51 olanzapine. 20 olmesartan. 26 olmesartn hydrochlorothiazide.29 olsalazine . 40 OMACOR. 28 omalizumab. 57 omedia. 35 omega-3 acid. 28 omeprazole.41 OMNICEF. 8 OMNI-PAC.8 ONCASPAR . 16 ondansetron. 20 ONTAK. 16 onxol. 16 OPHTHALMIC ANTIINFECTIVE CORTICOSTEROIDS . 54 OPHTHALMIC CORTICOSTEROID DRUGS. 54 OPHTHALMIC MEDICATIONS. 54 OPHTHALMIC TOPICAL ANTIBACTERIAL DRUGS. 55 oprelvekin. 44 ORAL ANTICOAGULANTS, VITAMIN K. 49 ORAL ANTIFUNGAL DRUGS.8 ORAL DERMATOLOGICAL DRUGS. 32 ORAL HYPOGLYCEMICS & COMBOS. 37 ORAP. 19 ORENCIA. 17 ORFADIN. 38 orphenadrine. 44 orphenadrine compound . 44 orphengesic, forte. 44 ORTHOCLONE . 17 oseltamivir. 10 OTHER ANTIARRHYTHMICS. 29 OTHER ANTICONVULSANTS . 23 OTHER ANTIDEPRESSANTS. 23!
Antihistamine. If symptoms redevelop, use of the decongestants could be repeated every couple of weeks. The value of desensitization, or "allergy shots, " is quite variable. This treatment is an effort to familiarize the immune system with the foreign material so that it stops over-reacting to it. The treatments may or may not offer long-term benefits, and tend to be more successful when given prior to puberty. Before pursuing this treatment, do some reading, and discuss the probability of benefit with your doctor. The prevention of life-threatening allergies calls for different strategies. The best approach, when the source of the allergy is known, is to avoid it. An example would be an antibiotic or a food. When the source may be unavoidable-such as a bee-traveling with an epi-pen is prudent precaution. Epinephrine is life-saving in a severe allergy attack, and the epipen is an injectable dose of epinephrine. The pen comes in adult and child doses. When exposures to the allergen the source of the allergy ; are likely to be recurrent, desensitization is generally indicated. I hope this advice helps you enjoy the spring and summer and
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PHARMACEUTICAL PREPARATIONS, VETERINARY THE BRITISH DRUG HOUSES, PREPARATIONS; SANITARY CHEMICAL ; SUBSTANCES, INFANTS LIMITED. ' AND INVALIDS' FOODS; DISINFECTANTS NOT INCLUDED IN OTHER CLASSES; PREPARATIONS FOR DESTROYING VERMIN; WEED EXTERMINATING AND DESTROYING PREPARATIONS.
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GASTROINTESTINAL AGENTS Antidiarrheals & Antispasmodics Drug Name bentyl CANTIL colytrol levbid levsin levsinex lomotil MOTOFEN PAMINE paregoric pro-banthine 15mg PRO-BANTHINE 7.5MG QUARZAN robinul forte SIMETYL SIMETYL ELIXiR Antiemetics Drug Name ALOXI antivert 15.5mg, 25mg tablet ANZEMET compazine COMPAZINE SYRUP EMEND KYTRIL MARINOL phenergan suppository TRANSDERM-SCOP ZOFRAN ZOFRAN ODT Generic Name palonosetron hcl meclizine hcl dolasetron mesylate prochlorperazine maleate prochlorperazine edisylate aprepitant granisetron hcl dronabinol promethazine hcl scopolamine hydrobromide onfansetron hcl ondansefron Drug Tier 4 1 4 Requirements Limits PA g ; PA Generic Name dicyclomine hcl mepenzolate bromide atrop sulf scopol hb hyoscy hyoscyamine sulfate hyoscyamine sulfate hyoscyamine sulfate diphenoxylate hcl atropine sulfate difenoxin hcl atropine sulfate scopolamine methylbromide paregoric propantheline bromide propantheline bromide clidinium bromide glycopyrrolate atrop sulf scopol hb hyoscy simethicone bellad alk Drug Tier 1 3 1 Requirements Limits g ; I g.
Nci ondansetron, a pilot study of intravenous ondansetron for and tacrolimus.
Animals. Male Swiss albino mice 2330 g ; from Morini San Polo d'Enza, Italy ; were used. The mice were housed 15 per cage. The cages were placed in the experimental room 24 hr before the test for adaptation. The animals were fed a standard laboratory diet and tap water ad libitum and kept at 23 1C with a 12-hr light dark cycle, light on at 7 A.M. All experiments were carried out according to the guidelines of the European Community Council for experimental animal care. Passive-avoidance test. The test was performed according to the step-through method described by Jarvik and Kopp 1967 ; . The apparatus consists of a two-compartment acrylic box with a lighted compartment connected to a darkened one by a guillotine door. Mice, as soon as they entered the dark compartment, received a punishing electrical shock 0.5 mA, 1 sec ; . The latency times for entering the dark compartment were measured in the training test and after 24 hr in the retention test. For memory disruption, mice were either exposed to a hypoxic environment 5%O2 in water-saturated nitrogen ; for 8 min up to 30 sec before passive avoidance training or i.p. injected with amnesic drugs. BIMU 1, BIMU 8, ondansetron, physostigmine and piracetam were injected 20 min before the training session while scopolamine, dicyclomine, GR 125487 and SDZ 205557 were injected immediately after termination of the training session. The maximum entry latency allowed in the retention session was 120 sec. The deficit in passive avoidance performance was expressed as the difference in seconds ; between retention and training latencies. Hole board test. The hole board test uses a 40 cm square plane with 16 flush-mounted cylindrical holes diameter 3 cm ; distributed 4 by 4 equidistant, grid-like manner. The mice were placed in the center of the board one by one and left to move about freely for a period of 5 min each. Two photoelectric beams, crossing the plane from mid-point to mid-point of opposite sides, thus dividing the plane into four equal quadrants, automatically signaled the movement of the animals on the surface of the plane. Miniature photoelectric cells, in each of the 16 holes, recorded the exploration of the holes head plunging activity ; by the mice. Rota rod test. The apparatus consists of a base platform and a rotating rod of 3-cm diameter with a non-skid surface. The rod was placed at a height of 15 cm from the base. The rod, 30 cm in length, was divided into five equal sections by six disks. Thus up to five mice were tested simultaneously on the apparatus, with a rod-rotation speed of 16 r.p.m. The integrity of motor coordination was assessed on the basis of the number of falls from the rod in 30 sec, according to Vaught et al. 1985 ; . Performance time was measured before and 15, 30 and 45 min after s.c. administration of the drugs. Spontaneous activity meter Animex ; . Locomotor activity in mice was quantified using an Animex activity meter Type S LKB, Farad, Sweden ; set to maximum sensitivity. Mice were placed on the top of the Animex activity meter and each movement produced a.
Donnie d , funny story bear with me ; my sis goes to mexico and buys their medicine.
If it is, i going to find a lawyer that , hopefully can make a case against either the prescribing doc, or the pharma.
Activating subscriptions document delivery linking to ingentaconnect alerting & rss feeds other library services keeping in touch register ondansetron is not superior to moderate dose metoclopramide in the prevention of post-operative nausea and vomiting after minor gynaecological surgery authors: monagle 1 ; barnes 1 ; goodchild 1 ; hewitt 1 source: european journal of anaesthesiology , volume 14, number 6, 1 november 1997 , pp.
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In animals, ondansetron reduces responses to static mechanical stimuli but is more effective against larger von frey forces than smaller ones and
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Ondansetron dosage and administration prevention of nausea and vomiting associated with highly emetogenic cancer chemotherapy the recommended adult oral dosage of ondansetron hydrochloride tablet is 24 mg given as three 8 mg tablets administered 30 minutes before the start of single-day highly emetogenic chemotherapy, including cisplatin ≥ 50 mg m 2.
Further Considerations In addition to identifying the highest risk factor scales and lowest protective factor scales, the prevention prioritization process may include several supplemental steps, such as: Compare county-level results to state-level results. Risk and protective factor scale scores from the statewide FYSAS are presented in Tables 13 and 14. A comparison to statewide results may reveal additional strengths and weaknesses in Miami -Dade County's risk and protective factor profile. For example, a risk factor scale that is not the most elevated within its domain may be designated as a target for prevention programming because it is notably higher in Miami -Dade County than across the state as a whole. Review the prevalence of ATOD use and other antisocial behaviors in your community. A high rate of alcohol use, for example, may dictate a different prevention strategy than a high rate of youth violence. The table on the second page in Appendix C provides a resource for this analysis by showing the behavioral outcomes that have Choosing Effective Prevention Strategies After completing the prioritization process and identifying key risk and protective factors for focused prevention efforts, the next step for communities is to select research-based, proven-effective programs that target these problem areas. A major breakthrough in the field of positive youth development in the past two decades has been the development and testing of programs, policies and practices that are shown to work to reduce adolescent drug use, violence, risky sexual behavior and school failure. State and national agencies have become increasingly interested in and committed to programs, policies and practices that have been rigorously tested for effectiveness. Prevention strategies identified as "tested, effective" are those that have been tested in well-controlled trials comparing schools, families, young people or communities that received the strategy with those that did not. Results of those trials showed that those who received the strategies were better off than those that did not, in terms of lower risk, greater protection and better behavioral outcomes.
ST Step Therapy ; : Step Therapy uses a patient's prior MDwise pharmacy claims history at any participating pharmacy to allow adjudication when there is a prior claim for a first-line agent within the specified timeframe. If there has been no prior claim for a first-line agent, then authorization is required PDL authorization forms found at mdwise ; . CT Concurrent Therapy ; : Concurrent Therapy uses a patient's prior MDwise pharmacy claims history to allow adjudication if there is a prior claim for the required concurrent agent. For example: If there has been no recent claim for the required concurrent agent, authorization is required. PA Prior Authorization ; : A drug with a designation of "PA" will require prior authorization by the Pharmacy Prior authorization departments. Please submit a MDwise "PA" form located at mdwise before the prescription is presented at a pharmacy. For questions regarding the prior authorization process, contact 800-558-1655. St. Catherine Hospital contracted prescribers, please contact 219-392-7090. Age Limit: An age edit allows claims for members within a defined age range to adjudicate without authorization. QLL Quantity Level Limits ; : Quantity Level Limits are limits on the specific quantity, days supply or prescriptions that can be dispensed within a given time frame. A normal 30 day supply is available under the pharmacy benefit unless listed otherwise. In the case of oral contraceptives, a 90 day supply is available without authorization. OTC Over the Counter ; : Requires a prescription; not available for Pkg C unless the drug is a first-line agent as noted on the PDL AB rated generics are mandatory when available.
Of 5-HTlA receptors in the dorsal hippocampus impairs acquisition and performance of a spatial task in a water maze. Brain Res 595: 50-56. Carli M, Tatarczynska E, Cervo L, Samanin R 1993 ; Stimulation of hippocampal 5-HT receptors causes amnesia and anxiolytic-like but not antidepressant-like effects in the rat. Eur J Pharmacol 234: 215221. Corradetti R, Ballerini L, Pugliese AM, Pepeu G 1992 ; Serotonin blocks the long-term potentiation induced by primed burst stimulation in the CA1 region of rat hippocampal slices. Neuroscience 465 1 l518. Costa11 B, Naylor RJ 1994 ; 5-HT, receptor antagonists in the treatment of cognitive disorders. In: 5-Hydroxytryptamine-3 receptor antagonists King FD, Jones BJ, Sanger GJ, eds ; , pp 203-219. Boca Raton, FL: CRC. Costa11 B, Coughlan J, Kelly ME, Naylor RJ, Tyres MB 1989 ; Scopolamine induced deficits in a T-maze reinforced alternation task are attenuated by 5-HT, receptor antagonists. Br J Pharmacol 98: 636P Crook T, Lakin M 1991 ; Effects of ondansetron in age-associated memory impairment. In: Biological psychiatry, Vol 2 Racagni G, et al., eds ; , pp 888-890. Amsterdam: Elsevier. Domeney AM, Costa11 B, Gerrard PA, Jones DNC, Naylor RJ, Tyers MB 1991 ; The effect of ondansetron on cognitive performance in the marmoset. Pharmacol Biochem Behav 38: 169-175. Freund TE Gulyas AI, Acsady L, Gores T, Toth K 1990 ; Serotonergic control of the hippocampus via local inhibitory interneurons. Proc Nat1 Acad Sci USA 87: 8501-8505. Jones DNC, Carey GJ, Costa11 B, Domeney AM, Gerrad PA, Naylor RJ, Tyres MB 1990 ; Scopolamine induced deficit in a primate object discrimination task are reversed by ondansetron GR 380 32F ; . Br J Pharmacol 100: 309P Huerta PT, Lisman JE 1993 ; Heightened synaptic plasticity of hippocampal CA1 neurons during a cholinergically induced rhythmic state. Nature 364: 723-725. Kilpatrick GJ, Tyers MB 1992 ; The pharmacological properties and functional roles of central 5-HT, receptors. In: Central and peripheral 5-HT, receptors Hamon H, ed ; , pp 33-57. London: Academic. Landfield PW 1976 ; Synchronous EEG rhythms: their nature and their possible functions in memory. In: Molecular and functional neurobiology Gispen WH, ed ; , pp 389424. Amsterdam: Elsevier. Larson J, Lynch G 1986 ; Induction of synaptic potentiation in hippocampus by patterned stimulation involves two events. Science 232: 985-988. Larson J, Wong D, Lynch G 1986 ; Patterned stimulation at the theta frequency is optimal for the induction of hippocampal long-term potentiation. Brain Res 386: 347-350. Maeda T, Kaneko S, Satoh M 1994 ; Inhibitory influence via 5-HT, receptors on the induction of LTP in mossy fiber-CA3 system of guinea-pig hippocampal slices. Neurosci Res 18: 277-282. Mott DD, Lewis DV 1991 ; Facilitation of the induction of long-term potentiation by GABA, receptors. Science 252: 1718-1720. Oleskevich S, Lacaille J-C 1992 ; Reduction of GABAb inhibitory postsynaptic potentials by serotonin via pre- and postsynaptic mech.
Dicp ann pharmacother 1990; 24 suppl ; : s51-s5 einhorn lh, nagy c, werner k, finn al ondansetron: a new antiemetic for patients receiving cisplatin chemotherapy.
They work by blocking the effects of the chemical dopamine in the body, but are only somewhat effective as antiemetic drugs.
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For more information on what ondansetron is used for, including possible off-label uses.
Ingredients Each suppository contains 16 milligrams of ondansetron. Your suppository also contains the following inactive ingredient: Witepsol S58. Zofran suppositories do not contain gluten or lactose. Manufacturer Your Zofran suppositories are supplied by: GlaxoSmithKline Australia Pty Ltd 1061 Mountain Highway Boronia Victoria 3155 Australia. Further Information Pharmaceutical companies are not in a position to give people an individual diagnosis or medical advice. Your doctor or pharmacist is the best person to give you advice on the treatment of your condition. You may also be able to find general information about your disease and its treatment from books, for example in public libraries. Do not throw this leaflet away. You may need to read it again. This leaflet was prepared on 16 January 2003. The information provided applies only to: Zofran Suppositories. Zofran Suppositories Issue No. 4.
As shown in Table 6, the three predictors of the increasing LOS in the recovery room, in decreasing order of importance, were: a longer duration of the procedure. older age of the patient. the antiemetic drug administered the use of granisetron or dolasetron versus ondansetron ; . Therefore, the variables that were not significant predictors of LOS both univariately and multivariately ; were as follows.
| Ondansetron iv glaxo nauseaDate: 11 24 04ISR Number: 4512413-8Report Type: Expedited 15-DaCompany Report #M2004-1695 Age: 29 YR Gender: Male I FU: I Outcome Dose Death 50 MG PRN, ORAL Clonidine 0.1 MG, PRN, ORAL Omeprazole Omeprazole ; 20 MG DAILY ORAL Temazepam Temazepam ; 20 MG PRN Voltarol Diclofenac ; 25 MG TID Diazepam Diazepam ; 10 MG PRN Heminevrin Clomethiazole ; Lorazepam Lorazepam ; 2.5 MG PRN Rohypnol Flunitrazepam ; 1 MG PRN Metoclopramide Metoclopramide ; 10 MG Q8H Buccastem Prochlorperazine ; Buscopan Hyoscine ; 10 MG, PRN Naltrexone Naltrexone ; Acupan Nefopam Hydrochloride ; Zofran Ondansetrln ; 8 MG, PRN SS SS SS ORAL SS ORAL PT Duration Foreign Body Aspiration Foreign Other Chlorpromazine PS ORAL Report Source Product Role Manufacturer Route.
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