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Sessor of a continuing professional development certificate but I know not whether I "practising". I do not dispense, an activity for which only a pharmacist may be responsible. We in industry do jobs which call upon our pharmacy education and knowledge but these jobs may also be done by non-pharmacists. Even the demanding role of a Qualified Person is not exclusively the domain of registered pharmacists. Will the Society please indicate for which of the sectors I should be putting myself forward? If it is the active sector and presuming the annual registration fee to be higher for active than for non-active folk, what will I be getting for my money? I do not find convincing the argument that membership fees for other professions allied to health are much greater. Why did ours rocket up by such a large amount.

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Main PROGRESS study, BP lowering was 5 2 mm with perindopril alone and there was no statistically significant beneficial effect on secondary prevention of stroke. Perindopril plus indapamide reduced BP by 12 Hg, and results were statistically highly significant in favor of the active treatment.9 In the study by Dufouil et al, 22 BP was reduced in the active treatment group by 12 8 Hg, suggesting that most, if not all, patients were receiving the ACE inhibitor and the diuretic; however, the authors do not provide us any of these important details. Extension of WMH that were present at baseline was not evaluated because this was a secondary prevention study and the object of the investigators was to prevent the appearance of new lesions. Given the anatomic basis of WMH, extension of preexisting WMH may represent new microvascular lesions and ischemia. Hippocampal atrophy has also been correlated with BP elevation and associated with cognitive impairment.15 Should hippocampal atrophy therefore have been evaluated? Another limitation of the study is the small number of subjects investigated, who may not be representative of the whole cohort. We cannot know whether the findings in the whole cohort with regard to stroke and dementia or impairment of cognition10 were reproduced in this small group of selected patients. Thus, most importantly, we are not provided with a clinical evaluation correlated with the neuroradiological imaging results. Was there a correlation between new WMH or, as mentioned above, increased volume of preexisting ones, and neurological deterioration or cognitive impairment? This would certainly make control of the progression of WMH with antihypertensive treatment matter very much to patients, practitioners, and health authorities. A final question relates to the specificity of the effect of the agents used. Was it the lowering of BP, the use of an ACE inhibitor any ACE inhibitor or specifically the one in the study ; , or the diuretic or the use of both in combination ; that resulted in these presumably favorable results? The study of Dufouil et al22 points the way to new studies that should examine in larger groups, to have adequate power, the result of treating patients with these or other antihypertensive agents on both imaging signs and cognitive and neurological results of treatment. In the meantime, the data obtained by Dufouil et al and previous studies provide us additional basis for not withholding the use of antihypertensive agents from older people with elevated BP to prevent recurrence of stroke and progression toward cognitive impairment.
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Aspect of the flap and, hence the medial aspect of the brow on its anterior surface, to its desired position. Depending on the plan, this will reposition the medial brow 3 to 8 inferiorly, and 1 to 3mm medially. The mid and lateral brow is repositioned as determined by patient preference. Most often, an "apex lateral slant" appearance is created through manipulation of the lateral aspect of the scalp flap. This position is secured by suturing the galea beneath the hair bearing skin over the tail of the brow to the deep temporal fascia. Using the incision and hairline registration marks placed earlier as references the anterior hairline is repositioned. This position is also secured with Mitek anchor scalp fixation to the skull. Results: 22 women average age - 45 years range 32 to 62 presented for correction of their brow position and shape after brow lift surgery. All brows were lowered and the brow apex shifted laterally. The medial brow was lowered 3 to 10 average 6 mm ; relative to the intercanthal line. The anterior hairline was lowered 5 to 18 average 12mm ; .The repositioning has remained stable over a 1 to year follow-up. No revisional surgery to further reposition the brows has been requested. 2 patients developed areas of alopecia in the posterior scalp flap. One of these patients had a 10x 25 mm area of alopecia excised and closed. Pre- and postoperative images of a patient who underwent the above described procedure are shown below. See Figure1 pre-op left, post-op reverse browlift right ; . Conclusion: Scalp advancement, repositioning, and fixation to the skull can effectively reposition and reshape the surgically elevated brow, for example, ramipril.

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While many prospective trials have investigated lowering blood pressure in subjects with kidney impairment, there have been relatively few in populations that are clearly identified as pre-ESRD, and the range of clinical outcomes is limited. Of the 12 identified reports, eight primarily investigated the ability of antihypertensive agents to lower blood pressure in pre-ESRD subjects. Two papers reported short- and long-term follow-up of the same investigational trial, which included death and initiation of kidney replacement therapy as clinical outcomes; 27, 28 one study reported echocardiographic measures of left ventricular geometry, 29 and another study retrospectively investigated the association between blood pressure and hospitalizations.25 A number of antihypertensive agents have been tested to determine their ability to lower blood pressure in pre-ESRD subjects. Some studies identified in the literature review were prospective clinical trials with a before after design and no parallel control group.3033 These studies enrolled relatively small numbers of subjects range 4-33 ; with advanced kidney impairment CrCl 30 mL min ; and hypertension. Two such studies, by Acchiardo et al.30 indapamide [a diuretic] ; and Weidmann et al.31 nitrendpine [a dihydropyridine calcium antagonist] ; , utilized a washout or placebo period with no other antihypertensive agents administered during the active phase. Two other studies, by Hammond et al.32 minoxidil [a vasodilator] ; and Toto et al.33 losartan [an angiotensin receptor antagonist] ; , allowed other antihypertensive agents if needed to control hypertension. Each of these investigations showed that blood pressure was significantly lowered after administration of the particular agent after a variable period of follow up range, 1-20 months ; . In each, the blood pressure after treatment was not lowered below 145 80 mmHg. Other studies utilized randomized controlled trial designs.25, 28, 29, 34-37 Shiigai et al.34 ACE inhibitor vs. beta blocker ; and Bianchi et al.35 ACE inhibitor vs. calcium antagonist ; randomized subjects to specific agents in head-to-head comparisons. In each study, additional blood pressure agents were to be added if necessary. Shiigai et al.34 compared enalapril and metoprolol in 36 pre-ESRD subjects; no differences in blood pressure were noted between the two groups. The group randomized to the ACE inhibitor exhibited a reduction in urinary protein excretion and a stabilization of decline in kidney function over 2 years compared to the group receiving beta blocker. The report by Bianchi et al.35 involved 16 pre-ESRD subjects randomized to enalapril or nicardipine. A significant reduction in blood pressure in each group was noted at 52 weeks. There was no difference in the degree of blood pressure reduction between the randomized groups. Level of kidney function remained stable in both groups; subjects receiving the ACE inhibitor exhibited a reduction in urinary albumin excretion. Three studies randomized subjects to a specific antihypertensive versus a variety of blood pressure agents. Kamper et al.27 studied the effects of an ACE inhibitor compared to "conventional antihypertensive therapy" in 70 pre-ESRD patients. Subjects taking the ACE inhibitor were also given conventional agents diuretics, beta blockers, vasodilators ; if needed to control blood pressure. The intervention was carried out for 2 years, at which time the investigators reported a lowering of blood pressure from study entry to study end within each group; no differences were noted and isoflavone.

Fulbright Fellowship Dr. Carver Nebbe was awarded a Fulbright Fellowship to spend nine months in Kazan, Russia September, 2006 through May of 2007. Working with his hosts in Kazan and with Dr. Richard Dobyns, Professor of Family Medicine and former Fulbright scholar, Dr. Nebbe wrote a proposal to work with the Internal Medicine and Family Medicine Residency Programs associated with Kazan State Medical University to teach the use of evidence based medicine in a clinical setting. Nebbe will also be working with medical students and has been asked to provide input on the development of the medical school's ethics curriculum. The development of this project began when Carver visited Russia for a month during his third year of residency while there on an international fellowship he received through Yale University. Kazan is located 500 miles almost due east of Moscow, is the capitol of the Republic of Tatarstan, and is the home of 1.2 million people. It's cold in the winter. For anybody who has interest in talking to Carver about the process of getting a Fulbright or is interested in what he will be doing there, he would be happy to discuss his project or any ideas for research or continuing partnership with Kazan State Medical University. Dr. Nebbe completed the five-year Family Medicine-Psychiatry Residency Program on June 30th. The PROGRESS study 2001 ; examined the effects of a blood-pressure-lowering in 6, 105 people mean age 6410 years ; with a history of stroke or transient ischaemic attack. Subjects were randomly assigned to active treatment n 3, 051 ; or placebo n 3, 054 ; . Active treatment involved the ACE inhibitor Perindopril 4mg daily with the addition of the diuretic Indpamide 2.5mg daily. Thirteen percent of participants in the active group and 12% in the placebo group had diabetes. The mean blood pressure of all participants was 147 86mmHg; and among those classified as hypertensive, the mean was 159 94mmHg and among those classified as nonhypertensive, the mean was 136 79mmHg. The primary outcome was fatal or nonfatal stroke, with a mean duration of follow-up of 3.9 years. Ten percent in the active group and 14% in the placebo group had a stroke RR reduction 28% [CI 17-38%], p 0.0001 ; . Active treatment also reduced the risk of total major vascular events 26% [CI 16-34] ; . Blood pressure was reduced by an overall average of 9.0 4.0mmHg among those on active treatment compared with those on placebo. In addition, when compared with placebo, active combination therapy achieved greater reduction in blood pressure 12.3 5.0mmHg ; than single-drug therapy 4.9 2.8mmHg ; and combination therapy reduced stroke risk by 43% CI 30-54 ; , while single therapy produced no significant reduction in the risk of stroke. In the SCOPE study SCOPE Study Investigators, 2002 ; , 4, 964 people aged 70-89 years ; with mild hypertension SBP 160-179mmHg and or DBP 90-99mmHg ; were randomised to receive either angiotensin 1-receptor blocker Candesartan Cilexetil or placebo. The primary endpoint was major cardiovascular events defined as a combined endpoint of cardiovascular death, non-fatal MI and non-fatal stroke. The secondary endpoints focused on the effects on cognitive function and dementia, total mortality, cardiovascular mortality, MI, stroke, renal function, hospitalisation and quality of life. There was a 28% reduction p 0.041 ; in non-fatal strokes in elderly people treated with Candesartan Cilexetil compared with placeb, o and a nonsignificant trend to reduced risk 11% risk reduction, p 0.19 ; of major cardiovascular events in the active treatment group. Lowering blood pressure was associated with maintained cognitive function, as measured by the MMSE. Candesartan Cilexetil was well tolerated in elderly people. Dahlof et al 1991 ; reported the effects of active treatment three -blockers and one diuretic ; and placebo on the frequency of cardiovascular events among 1, 627 elderly hypertensive people aged 70-84 years ; with SBP 180-230mmHg and or DBP 90120mmHg. Eight hundred and twelve people were randomly allocated to active treatment and 815 to placebo, and were followed up for 65 months. Primary endpoints were stroke, MI, and other cardiovascular death. A 19.5 8.1mmHg difference in blood pressure between treatment groups was observed. Active treatment had significantly fewer primary endpoints 58 v 94, p 0.0031 ; , lower morbidity and mortality from stroke 29 v 53, p 0.0081 ; , and lower total mortality compared with the placebo 36 v 63, p 0.0079 ; . Two recent studies have reported that ACE inhibitors exert additional beneficial effects and slow the decline in physical function. Participants in a retrospective cohort study Gambassi et al, 2000 ; had CHF and were taking either an ACE inhibitor n 4911 ; or Digoxin n 14890 ; . 22% of Digoxin users and 27% of ACE inhibitor users had diabetes. The study compared outcomes of ACE inhibitors and Digoxin on 1-year mortality, morbidity and physical function measured by ADL ; among older and isoniazid. A Board of Trustees, established through collective bargaining by labor unions and employers. Pursuant to the trust agreement under which it was created, it provides comprehensive healthcare benefits to participants who are employed under various collective bargaining agreements, along with their dependents and retirees based on the cumulative impact of Defendants' wrongful conduct as alleged herein and were damaged as a direct and foreseeable result of such conduct. 54. Plaintiff Painting Industry Insurance and Annuity Funds "PIIAF" ; , who filed Civil.

Stevenson M. Mexico eyes fake drugs on border. CBSNews Available at: : cbsnews stories 2004 08 10 worl d main635220.shtml Accessed 3 22 2005 and vasodilan. ON THE ABOVE DATE AND TIME R O WAS DISPATCHED TO THE INCIDENT LOCATION IN REFERENCE TO A DEATH INVESTIGATION. UPON MY ARRIVAL I SPOKE WITH LT. VANDEMARK, AND OFFICERS SCHRAMM AND DAVIERO OF THE MYRTLE BEACH FIRE DEPT WHO WERE ON SCENE. LT. VANDEMARK STATED THAT THEY HAD LOCATED A DECEASED PERSON IN THE UPSTAIRS BEDROOM OF THE RESIDENCE. EMS HAD ALREADY CHECKED THE BODY AND LEFT BEFORE MY ARRIVAL. I THEN CLEARED THE RESIDENCE, AND SECURED IT FOR FURTHER INVESTIGATION. I INTERVIEWED THE COMPLAINANT WHO IS A NEIGHBOR. THE COMPLAINANT HINES ; STATED HE RECEIVED A CALL FROM THE DECEASED SISTER WHO LIVES IN LONGS AT APPROXIMATELY 0930 HRS THIS DATE. THE SISTER STATED TO THE COMPLAINANT THAT THE DECEASED WAS TO BE AT WORK AT THE HAIR SALON BY 0900 HRS TODAY, AND HAD NOT SHOWN UP WHICH WAS NOT NORMAL. THE COMPLAINANT ADVISED HE SAW THAT THE DECEASED VEHICLE WAS STILL IN THE DRIVEWAY, AND THE POOL IN THE BACKYARD APPEARED TO BE ALMOST OVERFLOWING FROM THE WATER HOSE. THE COMPLAINANT ADVISED HE KNOCKED ON BOTH THE SIDE AND REAR DOOR BUT GOT NO RESPONSE. THE COMPLAINANT ADVISED HE COULD HEAR THE DOGS STILL UPSTAIRS IN THE RESIDENCE. THE COMPLAINANT ADVISED HE THEN USED HIS KEY TO THE RESIDENCE AND ENTERED TO CHECK ON THE DECEASED. HE STATED HE LOCATED THE DECEASED IN THE UPSTAIRS BEDROOM LAYING FACE DOWN BESIDE THE BED UNCONSCIOUS. CPL. GARDNER WAS CALLED TO THE SCENE, ALONG WITH DETECTIVES CURRY, COX, CRIME SCENE IANNONE, AND DEPUTY CORONER TAMARA WILLARD. THE NEXT OF KIN SISTER, DIANE SCHNEIDER ; - 843-333-3737 SIGNED A CONSENT TO SEARCH FOR US TO RE-ENTER THE RESIDENCE FOR FURTHER INVESTIGATION. ACCORDING TO THE SISTER, THE DECEASED HAD SEVERAL MEDICAL CONDITIONS TO INCLUDE BLOOD CLOTS IN THE AORTA AND LEG, AND WAS BEING TREATED BY DR. BRIAN ADLER IN SURFSIDE BEACH. THE DECEASED WAS ON THE FOLLOWING MEDICATIONS WHICH WERE TURNED OVER TO DEPUTY CORONER WILLARD. ALBUTEROL 90MCG FOLIC ACID 1MG CILOSTAZOL 100MG TOPROL XL 100 LEXAPRO 10MG VYTORIN 10 40 PROMETHAZINE 25MG NIASPAN 500MG INDAPAMIDE 2.5MG THE SCENE WAS PHOTOGRAPHED BY CRIME SCENE, AND MCMILIAN AND SMALL WAS CALLED BY DEPUTY CORONER WILLARD, WHO AUTHORIZED REMOVAL OF THE BODY.

The medicines listed are the generic or drug names, not the brand names Beta-blockers lower blood pressure by reducing the amount of a particular hormone a chemical messenger in the blood ; in the body, which is made by the kidneys and which increases blood pressure. Examples are: acebutolol, atenolol, bisoprolol, metoprolol, oxprenolol, propanolol, pindolol and timolol Diuretics work by increasing the amount of salt that your kidneys put out in your urine. This means that your body loses more salt, which brings out extra water with it. This reduces the amount of fluid around the cells and lowers your blood pressure. Examples are: bendroflumethiazide also called bendrofluazide ; , chlorothiazide, chlorthalidone, cyclopenthiazide, hydrochlorothiazide, indapaide ACE inhibitors work by reducing the amount of a hormone angiotensin II made by the kidneys. This hormone plays an important role in controlling blood pressure. Examples are: captopril, cilazapril, enalapril, fosinopril, lisinopril, perindopril, ramipril, trandolapril Calcium-channel blockers relax the arteries large blood vessels ; in your body and this lowers your blood pressure. Examples are: amlodipine, diltiazem, felodipine, isradipine, lacidipine, nicardipine nifedipine, nisoldipine, verapamil Statins Statins lower cholesterol levels in the blood by slowing down the production of cholesterol and by helping the liver to remove cholesterol that is already present in the blood stream. Examples are: atorvastatin, fluvastatin, pravastatin, rosuvastatin, simvastatin and ketorolac. F irst Draft provides members of the entertainment industry s creative community scribes, directors, development e xecutives science based health information services for comedy, drama, talk-shows, reality-based programming, movies made for television, feature films, and music videos. For free consultation on scripts and projects, please call toll free, because indspamide dose. 1. Wezler K, Boger A: [The dynamics of the arterial system]. Ergebnisse Physiologie 41: 292, 1939 O'Rourke MF, Kelly R, Avolio A: The Arterial Pulse. Philadelphia. London, Lea & Febiger, 1992 3. O'Rourke M: Arterial stiffness, systolic blood pressure, and logical treatment of arterial hypertension. Hypertension 15: 339 347, O'Rourke M: Mechanical principles in arterial disease. Hypertension 26: 29, 1995 Izzo JL Jr: Arterial stiffness and the systolic hypertension syndrome. Curr Opin Cardiol 19: 341352, 2004 Pannier BM, Guerin AP, Marchais SJ, London GM: Different aortic reflection wave responses following long-term angiotensin-converting enzyme inhibition and beta-blocker in essential hypertension. Clin Exp Pharmacol Physiol 28: 1074 1077, Dahlof B, Sever PS, Poulter NR, Wedel H, Beevers DG, Caulfield M, Collins R, Kjeldsen SE, Kristinsson A, McInnes GT, Mehlsen J, Nieminen M, O'Brien E, Ostergren J: Prevention of cardiovascular events with an antihypertensive regimen of amlodipine adding perindopril as required versus atenolol adding bendroflumethiazide as required, in the Anglo-Scandinavian Cardiac Outcomes Trial-Blood Pressure Lowering Arm ASCOT-BPLA ; : A multicentre randomised controlled trial. Lancet 366: 895906, 2005 Williams B, Lacy PS, Thom SM, Cruickshank K, Stanton A, Collier D, Hughes AD, Thurston H, O'Rourke M; CAFE Investigators; Anglo-Scandinavian Cardiac Outcomes Trial Investigators; CAFE Steering Committee and Writing Committee: Differential impact of blood pressure-lowering drugs on central aortic pressure and clinical outcomes: Principal results of the Conduit Artery Function Evaluation CAFE ; Study. Circulation, 113: 12131225, 2006 London GM, Marchais SJ, Guerin AP, Metivier F, Adda H: Arterial structure and function in end-stage renal disease. Nephrol Dial Transplant 17: 17131724, 2002 London GM: The concept of ventricular vascular coupling: Functional and structural alterations of the heart and arterial vessels go in parallel. Nephrol Dial Transplant 13: 250 253, Ting CT, Chou CY, Chang MS, Wang SP, Chiang BN, Yin FC: Arterial hemodynamics in human hypertension. Effects of adrenergic blockade. Circulation 84: 1049 1057, Schiffrin EL, Deng LY: Structure and function of resistance arteries of hypertensive patients treated with a beta-blocker or a calcium channel antagonist. J Hypertens 14: 12471255, 1996 London GM, Asmar RG, O'Rourke MF, Safar ME: Mechanism s ; of selective systolic blood pressure reduction after a low-dose combination of perindopril indapamied in hypertensive subjects: Comparison with atenolol. J Coll Cardiol 43: 9299, 2004 Wilkinson IB, MacCallum H, Flint L, Cockcroft JR, Newby DE, Webb DJ: The influence of heart rate on augmentation index and central arterial pressure in humans. J Physiol 525: 263270, 2000 de Luca N, Asmar RG, London GM, O'Rourke MF, Safar ME: Selective reduction of cardiac and ketotifen.
References: 1. The Medical Devices Agency. 1995 ; Infusion systems. Device Bull, MDA DB 9503. May. 2. Gatford, JD. 1998 ; Nursing calculations, 5th ediction. Churchill-Livingstone. Edinburgh, for example, pharmacology.
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You know i looking forward to a video eeg in a hospital setting where medications are lowered and regions of seizure focus are mapped.
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Peter Schneider1, Andr Rupp2, Renate Schneider1 and Eugen Diesch1 1 Central Institute of Mental Health, Department of Neuropsychology, University of Heidelberg, 2 Section of Biomagnetism, Department of Neurology, University of Heidelberg Introduction For a number of conditions e.g. cluster headaches, cervical dystonia, PTSD, schizophrenia ; morphological changes in cortical structures have been observed. Recently, tinnitus sufferers have been reported to show subcortical differences in gray matter density Mhlau et al., 2006 ; . Here we ask, whether tinnitus is accompanied by structural anomalies in auditory cortex. Methods Structural MRI images T1-weighted, Siemens Trio, 3-T ; were obtained from 41 tinnitus patients and 49 controls. 12 of the tinnitus patients and 33 of the controls were professional musicians, including orchestra musicians. Hearing status was determined for all participants. In tinnitus sufferers, subjective ratings of tinnitus severity and audiometric measures of tinnitus frequency and minimal masking level of the tinnitus were obtained. The MR data were processed using the BrainVoyager software Brain Innovation ; . Sagittal MRI slices were segmented along the Sylvian fissure to obtain 3D gray matter surface reconstructions of the individual auditory cortices. MR data were also analyzed using voxel based morphometry SPM2based VBM ; . Results The gray matter volumes of a medial-to-lateral sequence of cross-sectional slices of Heschl's gyrus were calculated. The mHG volume ipsilateral to the tinnitus ear was considerably smaller than the hmologuous contralateral volume. The ipsilateral reduction was observed both in musicians and non-musicians. However, mHG volume was about a factor of two larger in musicians. Overall, the gray matter volume asymmetry was largest for the postero-medial third of mHG. The results of individual morphometry analysis were corroborated by VBM-based group analysis. Tinnitus intrusiveness, but not the minimal masking level of the tinnitus, was smaller in affected musicans than in affected non-musicians. Conclusions Tinnitus sufferers demonstrated decreased gray matter volume of the primary audi92.
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CaOxSFs 112 ; 113 ; rather 114 ; beginswith whichcandissolvethe measuresfailing, d-penicillamine, -mercaptopropionylglycine, or captopril, can fever, proteinuria, serumsicknesstypereactions, andevenfranknephrotic Treatment trials: stone recurrence outcomes Todate, 7of8prospectivetrials, eachlastingatleast3years, idiopathicCaOxSFs Table5 ; .Table5makesclearthatdropout rateswerehigh, and despitetheseimperfections, Thiazide and citrate: recurrent calcium and2 115, 116 ; indicatetherapeuticbenefit Table5 ; .Thethird 117 ; lacksadouble-blinddesign, anditscontrolsubjectsformedfewer in3randomized, controlled, double-blindtrials.Chlorthalidone 118 ; atdosesof25mg dor50mg dgaveequivalentresultsthat arepooled Table5 ; equivalenttochlorthalidone 119 ; , asdidindapamide 120 ; . Dietary changes for recurrent calcium SFs.Borghietal. 32 ; Table5 ; .The2diets butby5years, the normalcalcium, reducedsodium, andproteindietprovedsuperior.

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Background Annual opium survey in Afghanistan The United Nations Office on Drugs and Crime UNODC ; has conducted an annual opium poppy survey in Afghanistan since 1994. The survey collects and analyses information on the location and extent of opium poppy cultivation, on the potential production of opium, as well as on opium prices in that country. The results provide a detailed picture of the current year's opium season and enable the identification of trends. This information is essential for planning, implementing and monitoring the impact of the measures required for tackling a problem which, in a country that has become by far the largest source of illicit opium and heroin trafficked in the world, has clearly assumed international dimensions. UNODC's opium survey in Afghanistan is implemented in the technical framework of its global Illicit Crop Monitoring Programme ICMP ; . The objective of ICMP is to assist the international community in monitoring the extent and evolution of illicit crops in the context of the elimination objective adopted at the General Assembly Special Session on Drugs in June 1998. The high level of opium poppy cultivation in Afghanistan During the 1990s, Afghanistan firmly established itself as the largest source of illicit opium and its derivative, heroin, in the world. By the end of the 1990s, Afghanistan provided about 70% of global illicit opium production, well ahead of Myanmar about 22% ; and Lao PDR about 3% ; . Primarily supplying countries in South West Asia, Central Asia, East and West Europe, as well as in South Asia, the Arabian peninsula and Africa, illicit opiates of Afghan origin were consumed by an and lozol.
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